Altered function of factor I caused by amyloid β: Implication for pathogenesis of age-related macular degeneration from drusen

Journal of Immunology

Volumn 181, Issue 1, 2008, Pages 712-720

  Wang, Jiying ^a   Ohno Matsui, Kyoko ^a   Yoshida, Takeshi ^a   Kojima, Ariko ^a   Shimada, Noriaki ^a   Nakahama, Ken Ichi ^a   Safranova, Olga ^a   Iwata, Nobuhisa ^b   Saido, Takaomi C ^b   Mochizuki, Manabu ^a   Morita, Ikuo ^a  

^a TOKYO MEDICAL AND DENTAL UNIVERSITY   (Japan)

^b INST OF PHYS AND CHEMICAL RESEARCH   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN; COMPLEMENT COMPONENT C3B; COMPLEMENT FACTOR H; COMPLEMENT FACTOR I; MEMBRANE METALLOENDOPEPTIDASE;

ANIMAL TISSUE; ARTICLE; COMPLEMENT ACTIVATION; COMPLEMENT SYSTEM; CONTROLLED STUDY; DRUSEN; INFLAMMATION; MOUSE; NONHUMAN; PATHOGENESIS; PIGMENT EPITHELIUM; PRIORITY JOURNAL; RETINA MACULA AGE RELATED DEGENERATION; CELL AGING; CELL LINE; GENE EXPRESSION REGULATION; GENETICS; HUMAN; METABOLISM; PHYSIOLOGY; PROTEIN BINDING; RETINA MACULA DEGENERATION;

AMYLOID BETA-PROTEIN; CELL AGING; CELL LINE; COMPLEMENT C3B; COMPLEMENT FACTOR H; COMPLEMENT FACTOR I; GENE EXPRESSION REGULATION; HUMANS; MACULAR DEGENERATION; NEPRILYSIN; PROTEIN BINDING; RETINAL DRUSEN;

EID: 47949105602     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.181.1.712     Document Type: Article

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