Effect of the knockdown of amyloid precursor protein on intracellular calcium increases in a neuronal cell line derived from the cerebral cortex of a trisomy 16 mouse

Experimental Neurology

Volumn 209, Issue 1, 2008, Pages 234-242

  Rojas, Guillermo ^a,b   Cárdenas, Ana María ^b   Fernández Olivares, Paola ^b   Shimahara, Takeshi ^c   Segura Aguilar, Juan ^a   Caviedes, Raúl ^a   Caviedes, Pablo ^a  

^a UNIVERSITY OF CHILE   (Chile)

^b UNIVERSIDAD DE VALPARAÍSO   (Chile)

^c CNRS   (France)

Author keywords

Antisense; Calcium; Down syndrome; Glutamate; Nicotine

Indexed keywords

AMYLOID PRECURSOR PROTEIN; CALCIUM; IONOMYCIN; MESSENGER RNA; NICOTINE; RNA;

ANIMAL CELL; ANIMAL MODEL; ARTICLE; BRAIN CORTEX; CALCIUM CELL LEVEL; CALCIUM HOMEOSTASIS; CELL MEMBRANE DEPOLARIZATION; GENETIC TRANSFECTION; MOUSE; NERVE CELL; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; TRISOMY 16;

AMYLOID BETA-PROTEIN PRECURSOR; ANIMALS; BLOTTING, WESTERN; CALCIUM; CELL LINE; CEREBRAL CORTEX; EXCITATORY AMINO ACID AGONISTS; IONOMYCIN; IONOPHORES; KINETICS; MICE; MICE, TRANSGENIC; NEURONS; NICOTINE; NICOTINIC AGONISTS; RECEPTORS, GLUTAMATE; RNA, ANTISENSE; TRISOMY;

EID: 37549007249     PISSN: 00144886     EISSN: 10902430     Source Type: Journal    
DOI: 10.1016/j.expneurol.2007.09.024     Document Type: Article

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