CCL19 is constitutively expressed in the CNS, up-regulated in neuroinflammation, active and also inactive multiple sclerosis lesions

Journal of Neuroimmunology

Volumn 190, Issue 1-2, 2007, Pages 72-79

  Krumbholz, M ^a,b   Theil, D ^b   Steinmeyer, F ^a,b   Cepok, S ^c   Hemmer, B ^c,d   Hofbauer, M ^b   Farina, C ^a,e   Derfuss, T ^a,b   Junker, A ^a,b   Arzberger, T ^b   Sinicina, I ^b   Hartle, C ^f   Newcombe, J ^f   Hohlfeld, R ^a,b   Meinl, E ^a,b  

^a MAX PLANCK INSTITUTE OF NEUROBIOLOGY   (Germany)

^b UNIVERSITY OF MUNICH   (Germany)

^c HEINRICH HEINE UNIVERSITY   (Germany)

^d TECHNICAL UNIVERSITY OF MUNICH   (Germany)

^e DEPARTMENT OF NEUROSURGERY   (Italy)

^f UNIVERSITY COLLEGE LONDON   (United Kingdom)

Author keywords

Chemokines; CSF; ELISA; Immunosurveillance; Multiple sclerosis; Taqman

Indexed keywords

CHEMOKINE RECEPTOR CCR7; IMMUNOGLOBULIN G; MACROPHAGE INFLAMMATORY PROTEIN 3BETA; SECONDARY LYMPHOID TISSUE CHEMOKINE;

ARTICLE; CELL LYSATE; CENTRAL NERVOUS SYSTEM; CEREBROSPINAL FLUID ANALYSIS; CONTROLLED STUDY; CORRELATION ANALYSIS; DISEASE COURSE; ENCEPHALITIS; ENZYME LINKED IMMUNOSORBENT ASSAY; HUMAN; HUMAN TISSUE; IMMUNE RESPONSE; IMMUNOGLOBULIN PRODUCTION; IMMUNOLOGICAL TOLERANCE; IMMUNOSURVEILLANCE; MULTIPLE SCLEROSIS; NEUROLOGIC DISEASE; PATHOPHYSIOLOGY; PRIORITY JOURNAL; PROTEIN EXPRESSION; RELAPSE; REMISSION; UPREGULATION;

EID: 35448986091     PISSN: 01655728     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.jneuroim.2007.07.024     Document Type: Article

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