Expression of disease-causing lamin A mutants impairs the formation of DNA repair foci

Journal of Cell Science

Volumn 119, Issue 13, 2006, Pages 2704-2714

  Manju, Kaliyaperumal ^a   Muralikrishna, Bhattiprolu ^a   Parnaik, Veena K ^a  

^a CENTRE FOR CELLULAR AND MOLECULAR BIOLOGY   (India)

Author keywords

ATR kinase; DNA repair; Emerin; Laminopathies; Nuclear lamina; Phosphorylated H2AX

Indexed keywords

ATR PROTEIN; CISPLATIN; COMPLEMENTARY DNA; EMERIN; GREEN FLUORESCENT PROTEIN; LAMIN A; MUTANT PROTEIN;

ARTICLE; CONTROLLED STUDY; DEGENERATIVE DISEASE; DNA DAMAGE; DNA DETERMINATION; DNA REPAIR; DNA SYNTHESIS; GENE EXPRESSION; GENE LOCATION; GENE MUTATION; GENETIC CODE; HUMAN; HUMAN CELL; IN VIVO STUDY; MOLECULAR INTERACTION; MUSCULAR DYSTROPHY; NUCLEAR LAMINA; PRIORITY JOURNAL; PROGERIA; PROTEIN AGGREGATION; PROTEIN ASSEMBLY; PROTEIN PHOSPHORYLATION; ULTRAVIOLET RADIATION; WILD TYPE;

CELL CYCLE PROTEINS; CISPLATIN; DNA DAMAGE; DNA REPAIR; DNA REPAIR-DEFICIENCY DISORDERS; HELA CELLS; HISTONES; HUMANS; LAMIN TYPE A; MEMBRANE PROTEINS; MUTANT PROTEINS; NUCLEAR PROTEINS; PROGERIA; PROTEIN-SERINE-THREONINE KINASES; THYMOPOIETINS; TISSUE DISTRIBUTION; ULTRAVIOLET RAYS;

ATAXIA TELANGIECTASIA;

EID: 33746883839     PISSN: 00219533     EISSN: None     Source Type: Journal    
DOI: 10.1242/jcs.03009     Document Type: Article

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