Defects in nuclear structure and function promote dilated cardiomyopathy in lamin A/C-deficient mice

Journal of Clinical Investigation

Volumn 113, Issue 3, 2004, Pages 357-369

  Nikolova, Vesna ^a   Leimena, Christiana ^a   McMahon, Aisling C ^a   Tan, Ju Chiat ^a   Chandar, Suchitra ^a   Jogia, Dilesh ^a   Kesteven, Scott H ^a   Michalicek, Jan ^a   Otway, Robyn ^a   Verheyen, Fons ^b   Rainer, Stephen ^c   Stewart, Colin L ^d   Martin, David ^a   Feneley, Michael P ^a   Fatkin, Diane ^a  

^a VICTOR CHANG CARDIAC RESEARCH INSTITUTE   (Australia)

^b MAASTRICHT UNIVERSITY   (Netherlands)

^c ST VINCENT S HOSPITAL   (Australia)

^d NATIONAL CANCER INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DESMIN; LAMIN A; LAMIN C; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA; STEROL REGULATORY ELEMENT BINDING PROTEIN 1;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CALCIUM SIGNALING; CONGESTIVE CARDIOMYOPATHY; CYTOSKELETON; DISEASE COURSE; DISEASE SEVERITY; ELECTRON MICROSCOPY; GENE; GENE ACTIVATION; GENE EXPRESSION; GENE FUNCTION; GENE MUTATION; GENE STRUCTURE; HEART CONTRACTION; HEART LEFT VENTRICLE; HEART VENTRICLE HYPERTROPHY; HETEROCHROMATIN; LMNA GENE; MOUSE; NONHUMAN; NUCLEAR LAMINA; PATHOPHYSIOLOGY; PRIORITY JOURNAL; PROTEIN PROTEIN INTERACTION; PROTEIN TRANSPORT; ANIMAL; APOPTOSIS; CELL NUCLEUS; ECHOCARDIOGRAPHY; ELECTROCARDIOGRAPHY; GENETICS; HEART MUSCLE; HEART MUSCLE CELL; PATHOLOGY; PHYSIOLOGY; TIME;

ANIMALS; APOPTOSIS; CARDIOMYOPATHY, DILATED; CELL NUCLEUS; ECHOCARDIOGRAPHY; ELECTROCARDIOGRAPHY; LAMIN TYPE A; MICE; MYOCARDIUM; MYOCYTES, CARDIAC; TIME FACTORS;

EID: 11144355499     PISSN: 00219738     EISSN: None     Source Type: Journal    
DOI: 10.1172/JCI200419448     Document Type: Article

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