Loss of primary cilia results in deregulated and unabated apical calcium entry in ARPKD collecting duct cells

American Journal of Physiology - Renal Physiology

Volumn 290, Issue 6, 2006, Pages

  Siroky, Brian J ^a   Ferguson, William B ^c   Fuson, Amanda L ^c   Xie, Yi ^c   Fintha, Attila ^c   Komlosi, Peter ^c   Yoder, Bradley K ^b   Schwiebert, Erik M ^a   Guay Woodford, Lisa M ^a   Bell, P Darwin ^a,c  

^a UNIVERSITY OF ALABAMA AT BIRMINGHAM   (United States)

^b UNIVERSITY OF ALABAMA AT BIRMINGHAM   (United States)

^c University of Alabama at Birmingham   (United States)

Author keywords

Autosomal recessive polycystic kidney disease; Ca2+ permeability; Oak Ridge Polycystic Kidney; Polycystin 2

Indexed keywords

CALCIUM; GADOLINIUM; MANGANESE; POLYCYSTIN 2;

ANIMAL CELL; ARTICLE; CALCIUM DEPLETION; CALCIUM TRANSPORT; CILIATED EPITHELIUM; CONTROLLED STUDY; ELECTROPHYSIOLOGY; FLUORESCENCE MICROSCOPY; KIDNEY COLLECTING TUBULE; KIDNEY POLYCYSTIC DISEASE; MOUSE; NONHUMAN; PHOTOCHEMICAL QUENCHING; PRIORITY JOURNAL; PROTEIN EXPRESSION;

EID: 33744809273     PISSN: 1931857X     EISSN: 15221466     Source Type: Journal    
DOI: 10.1152/ajprenal.00463.2005     Document Type: Article

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