JNK1 is required to preserve cardiac function in the early response to pressure overload

Biochemical and Biophysical Research Communications

Volumn 343, Issue 4, 2006, Pages 1060-1066

  Tachibana, Hideo ^a   Perrino, Cinzia ^a   Takaoka, Hideyuki ^a   Davis, Roger J ^b   Naga Prasad, Sathyamangla V ^a   Rockman, Howard A ^a  

^a DUKE UNIVERSITY MEDICAL CENTER   (United States)

^b UNIVERSITY OF MASSACHUSETTS MEDICAL SCHOOL   (United States)

Author keywords

Apoptosis; c Jun NH2 terminal kinase; Cardiac hypertrophy

Indexed keywords

MITOGEN ACTIVATED PROTEIN KINASE 12; MITOGEN ACTIVATED PROTEIN KINASE P38; STRESS ACTIVATED PROTEIN KINASE 1;

ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DETERIORATION; DISEASE COURSE; EXERCISE RECOVERY; GENE DELETION; HEART DISEASE; HEART FUNCTION; HEART HYPERTROPHY; HEART VENTRICLE OVERLOAD; INFLAMMATORY INFILTRATE; KNOCKOUT MOUSE; MOUSE; NICK END LABELING; NONHUMAN; PRIORITY JOURNAL; WILD TYPE;

ANIMALS; AORTA; APOPTOSIS; BLOOD PRESSURE; CARDIOMEGALY; CONSTRICTION, PATHOLOGIC; FEMALE; IN SITU NICK-END LABELING; MALE; MICE; MICE, KNOCKOUT; MITOGEN-ACTIVATED PROTEIN KINASE 10; MITOGEN-ACTIVATED PROTEIN KINASE 8; MITOGEN-ACTIVATED PROTEIN KINASE 9; MYOCARDIUM; PHENOTYPE;

ANIMALIA;

EID: 33646034318     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2006.03.065     Document Type: Article

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