ERK activation in arsenite-treated G1-enriched CL3 cells contributes to survival, DNA repair inhibition, and micronucleus formation

Toxicological Sciences

Volumn 89, Issue 1, 2006, Pages 164-172

  Li, Ju Pi ^a   Lin, Jin Ching ^b   Yang, Jia Ling ^a  

^a NATIONAL TSING HUA UNIVERSITY   (Taiwan)

^b TAICHUNG VETERANS GENERAL HOSPITAL   (Taiwan)

Author keywords

Arsenite; Cell synchronization; Genomic instability; MAPK; Nucleotide excision repair

Indexed keywords

ARSENIC TRIOXIDE; CYTOCHALASIN B; DNA; MITOGEN ACTIVATED PROTEIN KINASE 1; MITOGEN ACTIVATED PROTEIN KINASE 3;

ARTICLE; CANCER CELL; CELL CULTURE; CELL CYCLE; CELL CYCLE G1 PHASE; CELL PROLIFERATION; CELL SURVIVAL; CONTROLLED STUDY; CULTURE MEDIUM; CYTOTOXICITY; DNA REPAIR; EXCISION REPAIR; GENE ACTIVATION; GENOMIC INSTABILITY; HUMAN; HUMAN CELL; LUNG NON SMALL CELL CANCER; MALIGNANT NEOPLASTIC DISEASE; MICRONUCLEUS; SIGNAL TRANSDUCTION;

ARSENITES; CARCINOMA, NON-SMALL-CELL LUNG; CELL LINE, TUMOR; CELL SURVIVAL; DNA REPAIR; DNA, NEOPLASM; DOSE-RESPONSE RELATIONSHIP, DRUG; G1 PHASE; HUMANS; LUNG NEOPLASMS; MAP KINASE SIGNALING SYSTEM; MICRONUCLEI, CHROMOSOME-DEFECTIVE;

EID: 29544432386     PISSN: 10966080     EISSN: 10960929     Source Type: Journal    
DOI: 10.1093/toxsci/kfj004     Document Type: Article

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