Arsenite-induced Cdc25C degradation is through the KEN-box and ubiquitin-proteasome pathway

Proceedings of the National Academy of Sciences of the United States of America

Volumn 99, Issue 4, 2002, Pages 1990-1995

  Chen, Fei ^a   Zhang, Zhuo ^a   Bower, Jacquelyn ^a   Lu, Yongju ^a   Leonard, Stephen S ^a   Ding, Min ^a   Castranova, Vince ^a   Piwnica Worms, Helen ^b   Shi, Xianglin ^a  

^a NATIONAL INSTITUTE FOR OCCUPATIONAL SAFETY AND HEALTH   (United States)

^b WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ARSENIC TRIOXIDE; GENE PRODUCT; PROTEASOME; PROTEIN KEN; PROTEIN TYROSINE PHOSPHATASE; UBIQUITIN; UNCLASSIFIED DRUG;

ARTICLE; CARCINOGENESIS; CELL CYCLE G2 PHASE; CELL DIVISION; CELL PROLIFERATION; CONTROLLED STUDY; CYTOTOXICITY; GENE MUTATION; GENOTOXICITY; HEAVY METAL POISONING; HUMAN; HUMAN CELL; PRIORITY JOURNAL; PROTEIN DEGRADATION;

ACETYLCYSTEINE; AMINO ACID MOTIFS; ARSENITES; BLOTTING, WESTERN; CDC25 PHOSPHATASE; CELL CYCLE; CELL CYCLE PROTEINS; CELL LINE; DOSE-RESPONSE RELATIONSHIP, DRUG; DOWN-REGULATION; G0 PHASE; G1 PHASE; G2 PHASE; HUMANS; MITOSIS; MUTAGENESIS, SITE-DIRECTED; MUTATION; PEPTIDES; PHOSPHORYLATION; PRECIPITIN TESTS; SACCHAROMYCES CEREVISIAE PROTEINS; TIME FACTORS; UBIQUITIN;

EID: 0037133189     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.032428899     Document Type: Article

References (43)