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Oncogene

Volumn 21, Issue 27, 2002, Pages 4191-4199

NBS1 promotes ATM dependent hosphorylation events including those required for G1/S arrest

(5) Girard, Pierre Marie a Riballo, Enriqueta a Begg, Adrian C b Waugh, Alastair a Jeggo, Penny A a

a UNIVERSITY OF SUSSEX (United Kingdom)

b ANTONI VAN LEEUWENHOEK HOSPITAL (Netherlands)

Author keywords

ATM; Cell cycle checkpoint; Ionizing radiation; Kinase activity; NBS

Indexed keywords

ATM PROTEIN; PROTEIN; PROTEIN NBS 1; UNCLASSIFIED DRUG;

ARTICLE; CELL CYCLE G1 PHASE; CELL CYCLE S PHASE; CELL SURVIVAL; CONTROLLED STUDY; ENZYME ACTIVITY; HUMAN; HUMAN CELL; IONIZING RADIATION; NIJMEGEN BREAKAGE SYNDROME; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION;

ATAXIA TELANGIECTASIA; CELL CYCLE PROTEINS; CELLS, CULTURED; CHROMOSOME BREAKAGE; DNA-BINDING PROTEINS; DOSE-RESPONSE RELATIONSHIP, RADIATION; G1 PHASE; GENES, CDC; HISTONES; HUMANS; IMMUNOLOGIC DEFICIENCY SYNDROMES; NEOPLASTIC SYNDROMES, HEREDITARY; NUCLEAR PROTEINS; PHOSPHORYLATION; PROTEIN KINASES; PROTEIN PROCESSING, POST-TRANSLATIONAL; PROTEIN-SERINE-THREONINE KINASES; RADIATION TOLERANCE; S PHASE; SYNDROME; TUMOR SUPPRESSOR PROTEIN P53; TUMOR SUPPRESSOR PROTEINS;

ATAXIA; ATAXIA TELANGIECTASIA;

EID: 0037142619 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1205596 Document Type: Article

Times cited : (103)

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