Induction of apoptosis by vanilloid compounds does not require de novo gene transcription and activator protein 1 activity

Cell Growth and Differentiation

Volumn 9, Issue 3, 1998, Pages 277-286

  Macho, Antonio ^a   Blázquez, M Vaile ^a   Navas, Plácido ^b   Muñoz, Eduardo ^a  

^a Universidad de Cordoba   (Spain)

^b UNIVERSITY OF CÓRDOBA   (Spain)

Author keywords

[No Author keywords available]

Indexed keywords

CAPSAICIN; PHOSPHOTRANSFERASE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE; RESINIFERATOXIN; SYNAPSIN I;

APOPTOSIS; ARTICLE; DRUG EFFECT; ELECTRON TRANSPORT; ENZYME ACTIVATION; GENETIC TRANSCRIPTION; HUMAN; HUMAN CELL; LEUKEMIA CELL LINE; MEMBRANE POTENTIAL; MITOCHONDRIAL MEMBRANE; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

ACETYLCYSTEINE; APOPTOSIS; BINDING SITES; CA(2+)-CALMODULIN DEPENDENT PROTEIN KINASE; CAPSAICIN; CYCLOSPORINE; DITERPENES; DNA-BINDING PROTEINS; ENZYME ACTIVATION; ENZYME INHIBITORS; HUMANS; JNK MITOGEN-ACTIVATED PROTEIN KINASES; JURKAT CELLS; MEMBRANE POTENTIALS; MITOCHONDRIA; MITOGEN-ACTIVATED PROTEIN KINASES; NADH, NADPH OXIDOREDUCTASES; NFATC TRANSCRIPTION FACTORS; NUCLEAR PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2; REACTIVE OXYGEN SPECIES; TRANSCRIPTION FACTOR AP-1; TRANSCRIPTION FACTORS; TRANSCRIPTION, GENETIC; TUMOR SUPPRESSOR PROTEIN P53;

EUKARYOTA;

EID: 15144357925     PISSN: 10449523     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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