Late sodium channel openings underlying epileptiform activity are preferentially diminished by the anticonvulsant phenytoin

Journal of Neurophysiology

Volumn 77, Issue 6, 1997, Pages 3021-3034

  Segal, Michael M ^a,b   Douglas, Andrea F ^a  

^a BOSTON CHILDREN S HOSPITAL   (United States)

^b HARVARD MEDICAL SCHOOL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANTICONVULSIVE AGENT; PHENYTOIN; SODIUM CHANNEL;

ACTION POTENTIAL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CELL MEMBRANE DEPOLARIZATION; CELL MEMBRANE PERMEABILITY; CONCENTRATION RESPONSE; CONTROLLED STUDY; EPILEPSY; NEUROBLASTOMA; NEUROTRANSMITTER RELEASE; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEGRADATION; SYNAPTIC TRANSMISSION;

EID: 0030857906     PISSN: 00223077     EISSN: None     Source Type: Journal    
DOI: 10.1152/jn.1997.77.6.3021     Document Type: Article

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