Increased tau phosphorylation on mitogen-activated protein kinase consensus sites and cognitive decline in transgenic models for Alzheimer's disease and FTDP-17: Evidence for distinct molecular processes underlying tau abnormalities

Molecular and Cellular Biology

Volumn 25, Issue 1, 2005, Pages 278-293

  Lambourne, Sarah L ^a   Sellers, Lynda A ^a   Bush, Toby G ^a   Choudhury, Shewly K ^a   Emson, Piers C ^a   Suh, Yoo Hun ^b   Wilkinson, Lawrence S ^a  

^a BABRAHAM INSTITUTE   (United Kingdom)

^b Seoul National University   (South Korea)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID PRECURSOR PROTEIN; CYCLIN DEPENDENT KINASE; GLYCOGEN SYNTHASE KINASE 3ALPHA; GLYCOGEN SYNTHASE KINASE 3BETA; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE 1; STRESS ACTIVATED PROTEIN KINASE; TAU PROTEIN;

ALZHEIMER DISEASE; AMNESIA; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CARBOXY TERMINAL SEQUENCE; CHROMOSOME 17; COGNITIVE DEFECT; CONTROLLED STUDY; ENZYME ACTIVATION; FEMALE; FRONTOTEMPORAL DEMENTIA; GENE EXPRESSION; GENE MUTATION; HUMAN; INTERMETHOD COMPARISON; MALE; MOLECULAR DYNAMICS; MOUSE; NONHUMAN; PARKINSONISM; PRIORITY JOURNAL; PROTEIN CONTENT; PROTEIN DEFECT; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; STATISTICAL SIGNIFICANCE; TRANSGENE;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN; ANIMALS; BLOTTING, WESTERN; BRAIN; CELL MEMBRANE; CYCLIN-DEPENDENT KINASE 5; CYCLIN-DEPENDENT KINASES; DNA, COMPLEMENTARY; ENZYME-LINKED IMMUNOSORBENT ASSAY; GLYCOGEN SYNTHASE KINASE 3; GLYCOGEN SYNTHASE KINASES; HUMANS; IMAGE PROCESSING, COMPUTER-ASSISTED; IMMUNOHISTOCHEMISTRY; IN SITU HYBRIDIZATION; MAP KINASE SIGNALING SYSTEM; MEMBRANE PROTEINS; MICE; MICE, TRANSGENIC; MUTATION; PHOSPHORYLATION; PROSENCEPHALON; PROTEIN STRUCTURE, TERTIARY; RHOMBENCEPHALON; SIGNAL TRANSDUCTION; TAU PROTEINS; TIME FACTORS; TRANSGENES;

ANIMALIA;

EID: 11144249896     PISSN: 02707306     EISSN: None     Source Type: Journal    
DOI: 10.1128/MCB.25.1.278-293.2005     Document Type: Article

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