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Volumn 196, Issue 4, 2002, Pages 481-492
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CD4+ T cells from glutamic acid decarboxylase (GAD)65 -specific T cell receptor transgenic mice are not diabetogenic and can delay diabetes transfer
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Author keywords
Autoimmunity; CD152; IL 10; Mouse, NOD; T lymphocytes, regulatory
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Indexed keywords
CYTOTOXIC T LYMPHOCYTE ANTIGEN 4;
EPITOPE;
GAMMA INTERFERON;
GLUTAMATE DECARBOXYLASE;
GLUTAMATE DECARBOXYLASE 65;
INTERLEUKIN 10;
INTERLEUKIN 2;
T LYMPHOCYTE RECEPTOR;
TUMOR NECROSIS FACTOR ALPHA;
UNCLASSIFIED DRUG;
ANIMAL CELL;
ANIMAL MODEL;
ANIMAL TISSUE;
ANTIGEN EXPRESSION;
ARTICLE;
AUTOIMMUNITY;
CONTROLLED STUDY;
CYTOKINE PRODUCTION;
DIABETES MELLITUS;
DIABETOGENESIS;
INSULITIS;
LYMPHOCYTE PROLIFERATION;
MOUSE;
NONHUMAN;
PRIORITY JOURNAL;
SCID MOUSE;
SPLEEN CELL;
T LYMPHOCYTE;
T LYMPHOCYTE ACTIVATION;
TRANSGENIC MOUSE;
ANIMALS;
ANTIGENS, CD;
ANTIGENS, DIFFERENTIATION;
CD4-POSITIVE T-LYMPHOCYTES;
CD8-POSITIVE T-LYMPHOCYTES;
CELL DIVISION;
CYTOKINES;
DIABETES MELLITUS;
EPITOPES, T-LYMPHOCYTE;
GENE EXPRESSION;
GLUTAMATE DECARBOXYLASE;
IMMUNOCONJUGATES;
ISOENZYMES;
MICE;
MICE, INBRED NOD;
MICE, TRANSGENIC;
PEPTIDES;
RECEPTORS, ANTIGEN, T-CELL, ALPHA-BETA;
RECEPTORS, INTERLEUKIN-2;
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EID: 0037136283
PISSN: 00221007
EISSN: None
Source Type: Journal
DOI: 10.1084/jem.20011845 Document Type: Article |
Times cited : (77)
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References (60)
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