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Volumn 166, Issue 2, 2001, Pages 1352-1359

Th1 to Th2 cytokine shifts in nonobese diabetic mice: Sometimes an outcome, rather than the cause, of diabetes resistance elicited by immunostimulation

Author keywords

[No Author keywords available]

Indexed keywords

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; AUTOIMMUNITY; CELL COMPARTMENTALIZATION; CELL DEATH; CELL PROTECTION; CONTROLLED STUDY; CYTOKINE RELEASE; DISEASE RESISTANCE; IMMUNOSTIMULATION; INSULIN DEPENDENT DIABETES MELLITUS; NONHUMAN; OBESITY; OUTCOMES RESEARCH; PANCREAS ISLET BETA CELL; PRIORITY JOURNAL; RAT; T LYMPHOCYTE ACTIVATION;

EID: 0035863721     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: 10.4049/jimmunol.166.2.1352     Document Type: Article
Times cited : (154)

References (44)
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  • 2
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    • (1991) J. Immunol. , vol.147 , pp. 1222
    • Serreze, D.V.1    Leiter, E.H.2
  • 19
    • 0034653472 scopus 로고    scopus 로고
    • Induction of diabetes in nonobese diabetic mice by Th2 T cell clones from a TCR transgenic mouse
    • (2000) J. Immunol. , vol.164 , pp. 3072
    • Poulin, M.1    Haskins, K.2
  • 32
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    • Defective activation of T suppressor cell function in nonobese diabetic mice: Potential relation to cytokine deficiencies
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    • Serreze, D.V.1    Leiter, E.H.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.