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Volumn 21, Issue 22, 2002, Pages 3532-3540

AKT2 is frequently upregulated in HER-2/neu-positive breast cancers and may contribute to tumor aggressiveness by enhancing cell survival

(7) Bacus, Sarah S a Altomare, Deborah A b Lyass, Ljuba a Chin, Dot Mon a Farrell, Michael P a Gudkov, Andrei c Testa, Joseph R b

a Ventana Medical Systems (United States)

b FOX CHASE CANCER CENTER (United States)

c LERNER RESEARCH INSTITUTE (United States)

Author keywords

AKT; Apoptosis; Breast tumors; HER 2 neu; Hypoxia; Overexpression

Indexed keywords

2 MORPHOLINO 8 PHENYLCHROMONE; PHOSPHATIDYLINOSITOL 3 KINASE INHIBITOR; WORTMANNIN;

ADULT; AGED; APOPTOSIS; ARTICLE; BREAST CANCER; CANCER INVASION; CANCER SURVIVAL; CELL DEATH; CELL SURVIVAL; ENZYME ACTIVATION; FEMALE; GENE AMPLIFICATION; GENE FREQUENCY; GENE OVEREXPRESSION; HUMAN; HUMAN CELL; HYPOXIA; IMMUNOHISTOCHEMISTRY; MAJOR CLINICAL STUDY; ONCOGENE NEU; PRIORITY JOURNAL; RECEPTOR UPREGULATION;

1-PHOSPHATIDYLINOSITOL 3-KINASE; ANDROSTADIENES; APOPTOSIS; BREAST NEOPLASMS; CARCINOMA; CELL HYPOXIA; CELL SURVIVAL; CHROMONES; ENZYME INHIBITORS; FEMALE; HUMANS; MORPHOLINES; PROTEIN-SERINE-THREONINE KINASES; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-AKT; RECEPTOR, ERBB-2; TRANSFECTION; TUMOR CELLS, CULTURED; UP-REGULATION;

EID: 0037118575 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1205438 Document Type: Article

Times cited : (121)

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