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Volumn 99, Issue 3, 2002, Pages 1335-1340
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Loss of Pin1 function in the mouse causes phenotypes resembling cyclin D1-null phenotypes
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Author keywords
[No Author keywords available]
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Indexed keywords
CYCLIN D1;
GLUTATHIONE TRANSFERASE;
ISOMERASE;
PEPTIDYLPROLYL ISOMERASE PIN1;
UNCLASSIFIED DRUG;
ANIMAL CELL;
ARTICLE;
BREAST DEVELOPMENT;
BREAST EPITHELIUM;
CELL DIFFERENTIATION;
CONTROLLED STUDY;
ENZYME SPECIFICITY;
FEMALE;
IMMUNOHISTOCHEMISTRY;
ISOMERISM;
KNOCKOUT MOUSE;
MOUSE;
NONHUMAN;
PHENOTYPE;
PIGMENT EPITHELIUM;
PRIORITY JOURNAL;
PROTEIN CONFORMATION;
PROTEIN EXPRESSION;
PROTEIN PHOSPHORYLATION;
SIGNAL TRANSDUCTION;
STRUCTURE ACTIVITY RELATION;
TISSUE DISTRIBUTION;
ANIMALS;
ATROPHY;
BODY WEIGHT;
CELL DIVISION;
CYCLIN D1;
FEMALE;
MALE;
MAMMARY GLANDS, ANIMAL;
MICE;
MICE, KNOCKOUT;
PEPTIDYLPROLYL ISOMERASE;
PHENOTYPE;
PHOSPHORYLATION;
PREGNANCY;
RETINA;
TESTIS;
ANIMALIA;
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EID: 0037022355
PISSN: 00278424
EISSN: None
Source Type: Journal
DOI: 10.1073/pnas.032404099 Document Type: Article |
Times cited : (308)
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References (41)
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