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Volumn 26, Issue 1, 2002, Pages 106-114
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The mu-opioid receptor gene polymorphism (A118G) alters HPA axis activation induced by opioid receptor blockade
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Author keywords
Adrenocorticotropin; Cortisol; Naloxone; Neuroendocrine; Opioid receptor; Polymorphism
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Indexed keywords
BETA ENDORPHIN;
CORTICOTROPIN;
CORTICOTROPIN RELEASING FACTOR;
MU OPIATE RECEPTOR;
NALOXONE;
SEROTONIN TRANSPORTER;
ADULT;
ARTICLE;
BINDING AFFINITY;
BRAIN FUNCTION;
BRAIN NERVE CELL;
CONTROLLED STUDY;
CORTICOTROPIN RELEASE;
EXON;
GENETIC POLYMORPHISM;
GENOTYPE;
HORMONE RESPONSE;
HUMAN;
HUMAN EXPERIMENT;
HYPOTHALAMUS HYPOPHYSIS ADRENAL SYSTEM;
MALE;
MUTATION;
NORMAL HUMAN;
PRIORITY JOURNAL;
RECEPTOR BINDING;
RECEPTOR BLOCKING;
ADULT;
AMINO ACID SUBSTITUTION;
CARRIER PROTEINS;
DNA;
ELECTROPHORESIS, POLYACRYLAMIDE GEL;
HORMONES;
HUMANS;
HYPOTHALAMO-HYPOPHYSEAL SYSTEM;
MALE;
MEMBRANE GLYCOPROTEINS;
MEMBRANE TRANSPORT PROTEINS;
MUTATION;
NALOXONE;
NARCOTIC ANTAGONISTS;
NERVE TISSUE PROTEINS;
PERSONALITY;
POLYMORPHISM, GENETIC;
PROTEIN DENATURATION;
RECEPTORS, OPIOID;
RECEPTORS, OPIOID, MU;
REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION;
SEROTONIN PLASMA MEMBRANE TRANSPORT PROTEINS;
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EID: 0036149110
PISSN: 0893133X
EISSN: None
Source Type: Journal
DOI: 10.1016/S0893-133X(01)00294-9 Document Type: Article |
Times cited : (234)
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References (44)
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