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Volumn 194, Issue 12, 2001, Pages 1755-1766
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Blocking chemokine responsive to γ-2/interferon (IFN)-γ inducible protein and monokine induced by IFN-γ activity in vivo reduces the pathogenetic but not the antiviral potential of hepatitis B virus-specific cytotoxic T lymphocytes
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Author keywords
Chemokines; Hepatitis B virus; Infectious immunity virus; Liver
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Indexed keywords
ANTIBODY;
CHEMOKINE;
CHEMOKINE RESPONSIVE TO GAMMA 2;
GAMMA INTERFERON;
GAMMA INTERFERON INDUCIBLE PROTEIN 10;
MONOKINE;
MONOKINE INDUCED BY INTERFERON GAMMA;
UNCLASSIFIED DRUG;
ANIMAL EXPERIMENT;
ANTIVIRAL ACTIVITY;
ARTICLE;
CELL TRANSFER;
CONTROLLED STUDY;
CYTOKINE PRODUCTION;
CYTOTOXIC T LYMPHOCYTE;
DISEASE SEVERITY;
HEPATITIS B;
HEPATITIS B VIRUS;
IMMUNOTHERAPY;
IN VIVO STUDY;
LIVER DISEASE;
LIVER INJURY;
MONONUCLEAR CELL;
MOUSE;
NONHUMAN;
NUCLEOTIDE SEQUENCE;
PATHOGENICITY;
PRIORITY JOURNAL;
TRANSGENIC MOUSE;
VIRUS REPLICATION;
ANIMALS;
CYTOTOXICITY, IMMUNOLOGIC;
HEPATITIS B;
HEPATITIS B VIRUS;
INTERFERON TYPE II;
LIVER;
MICE;
MICE, TRANSGENIC;
MONOKINES;
T-LYMPHOCYTES, CYTOTOXIC;
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EID: 0035905411
PISSN: 00221007
EISSN: None
Source Type: Journal
DOI: 10.1084/jem.194.12.1755 Document Type: Article |
Times cited : (219)
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References (47)
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