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Oncogene

Volumn 20, Issue 22, 2001, Pages 2737-2748

Exploitation of a non-apoptotic caspase to regulate the abundance of the cdkI p27KIP1 in transformed lymphoid cells

(3) Frost, V a Al Mehairi, S a Sinclair, A J a

a UNIVERSITY OF SUSSEX (United Kingdom)

Author keywords

Apoptosis; Cell cycle; Lymphoma; Protease

Indexed keywords

BENZYLOXYCARBONYLVALYLALANYLASPARTYL FLUOROMETHYL KETONE; CASPASE; CASPASE 8; CASPASE INHIBITOR; CYCLIN DEPENDENT KINASE INHIBITOR; PROTEIN P27;

APOPTOSIS; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVE SITE; ENZYME INHIBITOR INTERACTION; ENZYME MECHANISM; ENZYME REGULATION; ENZYME SPECIFICITY; ENZYME SUBSTRATE COMPLEX; HUMAN; HUMAN CELL; LYMPHOCYTE TRANSFORMATION; LYMPHOID CELL; PRIORITY JOURNAL; PROTEIN EXPRESSION; STRUCTURE ACTIVITY RELATION;

AMINO ACID CHLOROMETHYL KETONES; APOPTOSIS; B-LYMPHOCYTES; CASPASE 8; CASPASE 9; CASPASES; CELL CYCLE; CELL CYCLE PROTEINS; CELL DIVISION; CELL LINE, TRANSFORMED; CYCLIN-DEPENDENT KINASE INHIBITOR P27; CYCLIN-DEPENDENT KINASES; HUMANS; JURKAT CELLS; MICROTUBULE-ASSOCIATED PROTEINS; PROTEIN BIOSYNTHESIS; TIME FACTORS; TRANSCRIPTION, GENETIC; TUMOR SUPPRESSOR PROTEINS; UP-REGULATION;

EID: 0035901969 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1204367 Document Type: Article

Times cited : (30)

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