Early formation of mature amyloid-β protein deposits in a mutant APP transgenic model depends on levels of Aβ1-42

Journal of Neuroscience Research

Volumn 66, Issue 4, 2001, Pages 573-582

  Rockenstein, Edward ^a   Mallory, Margaret ^a   Mante, Michael ^a   Sisk, Abyanné ^a   Masliaha, Eliezer ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

Alzheimer disease; Amyloid; Neurodegeneration; Synaptophysin; Transgenic

Indexed keywords

AMYLOID BETA PROTEIN; COMPLEMENTARY DNA; GENE PRODUCT; PROTEIN THY 1; SYNAPTOPHYSIN; UNCLASSIFIED DRUG;

ALZHEIMER DISEASE; ANIMAL CELL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; FRONTAL CORTEX; GENE CONTROL; GENE MUTATION; HIPPOCAMPUS; MOUSE; NEOCORTEX; NONHUMAN; NUCLEOTIDE SEQUENCE; OLFACTORY BULB; PLAQUE FORMING CELL; PRIORITY JOURNAL; PROTEIN DETERMINATION; PROTEIN EXPRESSION; THALAMUS; TRANSGENIC MOUSE;

AGING; ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN; AMYLOID BETA-PROTEIN PRECURSOR; ANIMALS; BRAIN; CONGO RED; DISEASE MODELS, ANIMAL; GENE EXPRESSION REGULATION, DEVELOPMENTAL; MICE; MICE, NEUROLOGIC MUTANTS; MICE, TRANSGENIC; MICROSCOPY, ELECTRON; MUTAGENESIS, INSERTIONAL; MUTATION; NEURITES; NEURONS; PEPTIDE FRAGMENTS; PRESYNAPTIC TERMINALS; PROMOTER REGIONS (GENETICS); SENILE PLAQUES; THIAZOLES;

EID: 0035889404     PISSN: 03604012     EISSN: None     Source Type: Journal    
DOI: 10.1002/jnr.1247     Document Type: Article

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