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Oncogene

Volumn 20, Issue 6, 2001, Pages 659-668

A transcriptional activation function of p53 is dispensable for and inhibitory of its apoptotic function

(5) Kokontis, John M a Wagner, Andrew J a O'leary, Maura b Liao, Shutsung a Hay, Nissim b

a UNIVERSITY OF CHICAGO (United States)

b UNIVERSITY OF ILLINOIS AT CHICAGO (United States)

Author keywords

Apoptosis; Cell cycle arrest; p53; Transactivation; Transrepression

Indexed keywords

DNA; PROTEIN BCL 2; PROTEIN P53;

ALLELE; APOPTOSIS; ARTICLE; CANCER CELL CULTURE; CELL CYCLE; CELL CYCLE G1 PHASE; CELL CYCLE G2 PHASE; CONTROLLED STUDY; DNA DAMAGE; GENE ACTIVATION; GENE EXPRESSION; GENE THERAPY; HUMAN; HUMAN CELL; POINT MUTATION; PRIORITY JOURNAL; TRANSCRIPTION INITIATION; TUMOR SUPPRESSOR GENE;

APOPTOSIS; BCL-2-ASSOCIATED X PROTEIN; CARCINOMA; CELL CYCLE; CYCLIN-DEPENDENT KINASE INHIBITOR P21; CYCLINS; GENE EXPRESSION REGULATION, NEOPLASTIC; GENES, BCL-2; HUMANS; MALE; MUTATION; PROSTATIC NEOPLASMS; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2; TRANS-ACTIVATORS; TUMOR SUPPRESSOR PROTEIN P53;

EID: 0035825595 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1204139 Document Type: Article

Times cited : (71)

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