Familial amyloid polyneuropathy: Receptor for advanced glycation end products-dependent triggering of neuronal inflammatory and apoptotic pathways

Journal of Neuroscience

Volumn 21, Issue 19, 2001, Pages 7576-7586

  Sousa, Mónica Mendes ^a   Yan, Shi Du ^b   Fernandas, Rui ^a   Guimarães, António ^c   Stern, David ^b   Saraiva, Maria João ^a,d,e  

^a Teachers College   (United States)

^b Hospital Geral de Santo António ^*  (United States)

^c HOSPITAL DE SANTO ANTÓNIO   (Portugal)

^d UNIVERSITY OF PORTO   (Portugal)

^e UNIVERSITY OF PORTO   (Portugal)

Author keywords

Amyloid; Caspase 3; Familial amyloidotic polyneuropathy; Inducible nitric oxide synthase; Inflammatory cytokine; RAGE; Transthyretin

Indexed keywords

ADVANCED GLYCATION END PRODUCT RECEPTOR; AMYLOID; CASPASE 3; CYTOKINE; INTERLEUKIN 1BETA; NITRIC OXIDE SYNTHASE; PREALBUMIN;

AMYLOID NEUROPATHY; ANIMAL CELL; APOPTOSIS; ARTICLE; CELL DEATH; CLINICAL ARTICLE; CONTROLLED STUDY; ENDOTHELIUM CELL; FAMILIAL AMYLOID POLYNEUROPATHY; HUMAN; HUMAN TISSUE; IMMUNOHISTOLOGY; IN SITU HYBRIDIZATION; NERVE CELL NECROSIS; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN GLYCOSYLATION; RAT; SCHWANN CELL; SIGNAL TRANSDUCTION; SURAL NERVE;

EID: 0035478665     PISSN: 02706474     EISSN: None     Source Type: Journal    
DOI: 10.1523/jneurosci.21-19-07576.2001     Document Type: Article

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