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Volumn 29, Issue 15, 2001, Pages 3172-3180
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Cleavage of the Bloom's syndrome gene product during apoptosis by caspase-3 results in an impaired interaction with topoisomerase IIIα
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Author keywords
[No Author keywords available]
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Indexed keywords
CASPASE 3;
CELL PROTEIN;
DNA;
DNA TOPOISOMERASE;
GENE PRODUCT;
HELICASE;
PROTEINASE;
AMINO TERMINAL SEQUENCE;
APOPTOSIS;
ARTICLE;
AUTOSOMAL DISORDER;
BLOOM SYNDROME;
CANCER SUSCEPTIBILITY;
CONTROLLED STUDY;
DNA DAMAGE;
DNA REPAIR;
DNA REPLICATION;
ENZYME ACTIVATION;
ENZYME ACTIVITY;
ENZYME ANALYSIS;
ENZYME DEGRADATION;
ENZYME STRUCTURE;
EUKARYOTE;
GENE LOCATION;
GENE MUTATION;
GENE REPLICATION;
GENETIC ANALYSIS;
GENETIC RECOMBINATION;
GENETIC STABILITY;
HUMAN;
HUMAN CELL;
PRIORITY JOURNAL;
PROTEIN ANALYSIS;
PROTEIN DEGRADATION;
PROTEIN DOMAIN;
PROTEIN INTERACTION;
SIGNAL TRANSDUCTION;
STRESS;
STRUCTURE ANALYSIS;
ADENOSINE TRIPHOSPHATASES;
APOPTOSIS;
BLOOM SYNDROME;
CASPASE 3;
CASPASES;
CYCLOHEXIMIDE;
DNA HELICASES;
DNA TOPOISOMERASES, TYPE I;
ETOPOSIDE;
HELA CELLS;
HL-60 CELLS;
HUMANS;
MODELS, BIOLOGICAL;
MOLECULAR WEIGHT;
PROTEIN BINDING;
PROTEIN PROCESSING, POST-TRANSLATIONAL;
PROTEIN STRUCTURE, TERTIARY;
TUMOR NECROSIS FACTOR-ALPHA;
EUKARYOTA;
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EID: 0035419799
PISSN: 03051048
EISSN: None
Source Type: Journal
DOI: None Document Type: Article |
Times cited : (28)
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References (43)
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