Both caspase-dependent and caspase-independent pathways may be involved in hippocampal CA1 neuronal death because of loss of cytochrome c from mitochondria in a rat forebrain ischemia model

Journal of Cerebral Blood Flow and Metabolism

Volumn 21, Issue 5, 2001, Pages 529-540

  Zhan, Ren Zhi ^a   Wu, Chaoran ^a   Fujihara, Hideyoshi ^a   Taga, Kiichiro ^a   Qi, Sihua ^a   Naito, Makoto ^a   Shimoji, Koki ^a  

^a NIIGATA UNIVERSITY   (Japan)

Author keywords

Apoptosis; Caspases; Cycloheximide; Cytochrome c; DNA fragmentation; Hippocampus; Ischemic preconditioning; Mitochondria

Indexed keywords

CASPASE; CASPASE INHIBITOR; CYCLOHEXIMIDE; CYTOCHROME C; DNA FRAGMENT; N BENZYLOXYCARBONYLASPARTYL(2 METHYL)GLUTAMYL(2 METHYL)VALYLASPARTYL(2 METHYL)FLUOROMETHYLKETONE; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN ISCHEMIA; CAROTID ARTERY OBSTRUCTION; CONTROLLED STUDY; DENTATE GYRUS; ENZYME RELEASE; FOREBRAIN; HIPPOCAMPUS; HYPOTENSION; MALE; MITOCHONDRION; NERVE CELL NECROSIS; NICK END LABELING; NONHUMAN; PRIORITY JOURNAL; RAT; REPERFUSION;

EID: 0035012926     PISSN: 0271678X     EISSN: None     Source Type: Journal    
DOI: 10.1097/00004647-200105000-00007     Document Type: Article

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