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Volumn 20, Issue 15, 2000, Pages 5715-5723
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Delayed mitochondrial dysfunction in excitotoxic neuron death: Cytochrome c release and a secondary increase in superoxide production
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Author keywords
Apoptosis; Cytochrome c; Excitotoxicity; Glutamate; Mitochondria; NMDA; Reactive oxygen species; Respiratory chain; Superoxide
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Indexed keywords
ANTIMYCIN A1;
CASPASE;
CYTOCHROME C;
GLUTAMATE RECEPTOR AGONIST;
GLUTAMIC ACID;
N METHYL DEXTRO ASPARTIC ACID;
OLIGOMYCIN;
REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE);
ROTENONE;
STAUROSPORINE;
SUPEROXIDE;
SUPEROXIDE DISMUTASE;
UBIQUINOL CYTOCHROME C REDUCTASE;
ANIMAL CELL;
APOPTOSIS;
ARTICLE;
CONTROLLED STUDY;
ENZYME ACTIVATION;
ENZYME INHIBITION;
HIPPOCAMPUS;
HUMAN;
HUMAN CELL;
MEDULLOBLASTOMA;
MITOCHONDRIAL MEMBRANE;
MITOCHONDRIAL RESPIRATION;
NERVE CELL NECROSIS;
NONHUMAN;
OXIDATIVE STRESS;
PATHOGENESIS;
PRIORITY JOURNAL;
RAT;
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EID: 0034254932
PISSN: 02706474
EISSN: None
Source Type: Journal
DOI: 10.1523/jneurosci.20-15-05715.2000 Document Type: Article |
Times cited : (212)
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References (60)
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