Neuronal NOS inhibitor that reduces oxidative DNA lesions and neuronal sensitivity increases the expression of intact c-fos transcripts after brain injury

Journal of Biomedical Science

Volumn 8, Issue 4, 2001, Pages 336-341

  Cui, J ^a   Liu, P K ^a  

^a BAYLOR COLLEGE OF MEDICINE   (United States)

Author keywords

Gene expression; Neuroregeneration; Oxidative stress; Stroke; Transcription

Indexed keywords

3 BROMO 7 NITROINDAZOLE; INDAZOLE DERIVATIVE; MESSENGER RNA; NITRIC OXIDE; NITRIC OXIDE SYNTHASE INHIBITOR; PROTEIN C FOS; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN CORTEX; BRAIN INJURY; BRAIN ISCHEMIA; CELL DEATH; CELL REGENERATION; CONTROLLED STUDY; DNA DAMAGE; GENE EXPRESSION; GENETIC TRANSCRIPTION; NERVE CELL; NONHUMAN; ONCOGENE C FOS; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT; RAT STRAIN; REPERFUSION INJURY; RIBONUCLEASE PROTECTION ASSAY; STROKE;

ANIMALS; BRAIN INJURIES; DNA DAMAGE; ENZYME INHIBITORS; IN SITU HYBRIDIZATION; MALE; NEURONS; NITRIC OXIDE SYNTHASE; NITRIC OXIDE SYNTHASE TYPE I; OXIDATIVE STRESS; PROTO-ONCOGENE PROTEINS C-FOS; RATS; RATS, LONG-EVANS; RNA, MESSENGER;

EID: 0034917703     PISSN: 10217770     EISSN: 14230127     Source Type: Journal    
DOI: 10.1159/000054052     Document Type: Article

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