Oxidative damage to the c-fos gene and reduction of its transcription after focal cerebral ischemia

Journal of Neurochemistry

Volumn 73, Issue 3, 1999, Pages 1164-1174

  Cui, Jiankun ^a   Holmes, Eric H ^b   Liu, Philip K ^a  

^a BAYLOR COLLEGE OF MEDICINE   (United States)

^b PACIFIC NORTHWEST RESEARCH INSTITUTE   (United States)

Author keywords

Apoptosis; DNA repair; Gene expression; Neuroregeneration; Oxidative stress; Stroke

Indexed keywords

NITRIC OXIDE SYNTHASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ATTENUATION; BRAIN INJURY; BRAIN ISCHEMIA; CONTROLLED STUDY; DNA DAMAGE; GENETIC TRANSCRIPTION; MALE; NONHUMAN; ONCOGENE C FOS; OXIDATION; PRIORITY JOURNAL; RAT; REPERFUSION; RNA TRANSCRIPTION; TRANSCRIPTION REGULATION;

ANIMALS; DEOXYGUANOSINE; DNA REPAIR; GENE EXPRESSION REGULATION; GENES, FOS; IMMUNOHISTOCHEMISTRY; IN SITU HYBRIDIZATION; ISCHEMIC ATTACK, TRANSIENT; MALE; OXIDATIVE STRESS; RATS; RATS, LONG-EVANS; REPERFUSION INJURY; RNA, MESSENGER;

EID: 0032866416     PISSN: 00223042     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1471-4159.1999.0731164.x     Document Type: Article

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