A role for the p38 mitogen-activated protein kinase pathway in the transcriptional activation of p53 on genotoxic stress by chemotherapeutic agents

Cancer Research

Volumn 60, Issue 9, 2000, Pages 2464-2472

  Sanchez Prieto, Ricardo ^a   Rojas, Jose M ^a,c   Taya, Yoichi ^b   Gutkind, J Silvio ^a  

^a NATIONAL INSTITUTE OF DENTAL AND CRANIOFACIAL RESEARCH   (United States)

^b NATIONAL CANCER CENTER RESEARCH INSTITUTE   (Japan)

^c Instituto de Salud   (Spain)

Author keywords

[No Author keywords available]

Indexed keywords

CISPLATIN; DOXORUBICIN; MITOGEN ACTIVATED PROTEIN KINASE; PROTEIN P53;

ANIMAL CELL; APOPTOSIS; ARTICLE; CANCER CHEMOTHERAPY; CELL SURVIVAL; DNA DAMAGE; ENZYME ANALYSIS; ENZYME PHOSPHORYLATION; GENOTOXICITY; MOUSE; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; TRANSCRIPTION REGULATION;

3T3 CELLS; ANIMALS; ANTINEOPLASTIC AGENTS; APOPTOSIS; BLOTTING, WESTERN; CISPLATIN; DNA DAMAGE; DOSE-RESPONSE RELATIONSHIP, DRUG; DOXORUBICIN; FLOW CYTOMETRY; FLUORESCENT ANTIBODY TECHNIQUE; GENES, REPORTER; MICE; MITOGEN-ACTIVATED PROTEIN KINASES; P38 MITOGEN-ACTIVATED PROTEIN KINASES; PHOSPHORYLATION; PLASMIDS; STRESS; TIME FACTORS; TRANS-ACTIVATION (GENETICS); TRANSFECTION; TUMOR SUPPRESSOR PROTEIN P53;

EID: 0034193343     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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