Relevance of phosphorylation and truncation of tau to the etiopathogenesis of Alzheimer's disease

Frontiers in Aging Neuroscience

Volumn 10, Issue FEB, 2018, Pages

  Zhou, Yan ^a,b,c   Shi, Jianhua ^a,b   Chu, Dandan ^a   Hu, Wen ^a,b   Guan, Zongyu ^a   Gong, Cheng Xin ^b   Iqbal, Khalid ^b   Liu, Fei ^a,b  

^a NANTONG UNIVERSITY   (China)

^b NEW YORK STATE INSTITUTE FOR BASIC RESEARCH IN DEVELOPMENTAL DISABILITIES   (United States)

^c NANTONG MEDICAL COLLEGE   (China)

Author keywords

Alzheimer's disease; Hyperphosphorylation; Tau; Tau pathogenesis; Truncation

Indexed keywords

ASPARTIC ACID; EPITOPE; SERINE; TAU PROTEIN; THREONINE;

AGED; ALZHEIMER DISEASE; AMINO TERMINAL SEQUENCE; ANTIBODY LABELING; ARTICLE; BRAIN HOMOGENATE; CARBOXY TERMINAL SEQUENCE; CONTROLLED STUDY; FEMALE; HUMAN; HUMAN TISSUE; MALE; PATHOGENESIS; PROTEIN AGGREGATION; PROTEIN PHOSPHORYLATION; WESTERN BLOTTING;

EID: 85042407518     PISSN: None     EISSN: 16634365     Source Type: Journal    
DOI: 10.3389/fnagi.2018.00027     Document Type: Article

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