α-Synuclein promotes dilation of the exocytotic fusion pore

Nature Neuroscience

Volumn 20, Issue 5, 2017, Pages 681-689

  Logan, Todd ^a   Bendor, Jacob ^a   Toupin, Chantal ^a   Thorn, Kurt ^a   Edwards, Robert H ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALPHA SYNUCLEIN; BAFILOMYCIN; BETA SYNUCLEIN; BRAIN DERIVED NEUROTROPHIC FACTOR; GREEN FLUORESCENT PROTEIN; PROTON PUMP INHIBITOR; SECRETOGRANIN II; ISOPROTEIN;

ADRENAL CHROMAFFIN CELL; ANIMAL CELL; ARTICLE; CALCIUM TRANSPORT; CHROMAFFIN CELL; CHROMAFFIN GRANULE; CONTROLLED STUDY; GENE LOCATION; GENE MUTATION; GENE OVEREXPRESSION; GENETIC PREDISPOSITION; GENETIC TRANSFECTION; IMMUNOBLOTTING; IMMUNOFLUORESCENCE; INHIBITION KINETICS; LENTIVIRINAE; MOUSE; NERVE CELL CULTURE; NERVE ENDING; NEUROTRANSMITTER RELEASE; NONHUMAN; PARKINSON DISEASE; PRIORITY JOURNAL; SENSITIVITY AND SPECIFICITY; SYNAPSE VESICLE; TOTAL INTERNAL REFLECTION FLUORESCENCE MICROSCOPY; ANIMAL; BIOSYNTHESIS; CELL CULTURE; DILATATION; EXOCYTOSIS; GENETICS; HUMAN; KINETICS; KNOCKOUT MOUSE; METABOLISM; MUTATION; NERVE CELL; PATHOPHYSIOLOGY; PHYSIOLOGY; PRIMARY CELL CULTURE; SECRETORY VESICLE;

ALPHA-SYNUCLEIN; ANIMALS; CELLS, CULTURED; CHROMAFFIN CELLS; DILATATION; EXOCYTOSIS; HUMANS; KINETICS; MICE; MICE, KNOCKOUT; MUTATION; NEURONS; PARKINSON DISEASE; PRIMARY CELL CULTURE; PROTEIN ISOFORMS; SECRETORY VESICLES; SYNAPTIC VESICLES;

EID: 85015043895     PISSN: 10976256     EISSN: 15461726     Source Type: Journal    
DOI: 10.1038/nn.4529     Document Type: Article

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