Resistance to pathologic cardiac hypertrophy and reduced expression of CaV1.2 in Trpc3-depleted mice

Molecular and Cellular Biochemistry

Volumn 421, Issue 1-2, 2016, Pages 55-65

  Han, Jung Woo ^a   Lee, Young Ho ^a   Yoen, Su In ^a   Abramowitz, Joel ^b   Birnbaumer, Lutz ^b   Lee, Min Goo ^a   Kim, Joo Young ^a  

^a Yonsei University College of Medicine   (South Korea)

^b NATIONAL INSTITUTE OF ENVIRONMENTAL HEALTH SCIENCES   (United States)

Author keywords

Ca2+ influx; L type Ca2+ channel; Pathologic cardiac hypertrophy; Transient receptor potential canonical channels 3

Indexed keywords

BIOLOGICAL MARKER; CALCINEURIN; CALCIUM; CALCIUM CHANNEL CAV1.2; CALMODULIN; MESSENGER RNA; NEUROHORMONE; PHENYLEPHRINE; TRANSCRIPTION FACTOR NFAT; TRANSIENT RECEPTOR POTENTIAL CHANNEL 3; UNCLASSIFIED DRUG; VOLTAGE GATED CALCIUM CHANNEL; CACNA1C PROTEIN, MOUSE; CALCIUM CHANNEL L TYPE; TRANSIENT RECEPTOR POTENTIAL CHANNEL C;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CALCIUM CELL LEVEL; CALCIUM TRANSPORT; CARDIAC MUSCLE CELL; CONTROLLED STUDY; DEATH; GENE; GENE DELETION; GENE EXPRESSION; HEART VENTRICLE HYPERTROPHY; MALE; MESENTERIC ARTERY; MOUSE; NEWBORN; NONHUMAN; PATHOGENESIS; PROTEIN EXPRESSION; SYSTOLIC BLOOD PRESSURE; TRPC3 GENE; VASOCONSTRICTION; ANIMAL; BIOSYNTHESIS; CALCIUM SIGNALING; CARDIAC MUSCLE; CARDIOMEGALY; DEFICIENCY; GENE EXPRESSION REGULATION; GENETICS; KNOCKOUT MOUSE; METABOLISM; PATHOLOGY;

ANIMALS; CALCIUM CHANNELS, L-TYPE; CALCIUM SIGNALING; CARDIOMEGALY; GENE EXPRESSION REGULATION; MESENTERIC ARTERIES; MICE; MICE, KNOCKOUT; MYOCARDIUM; PHENYLEPHRINE; TRPC CATION CHANNELS; VASOCONSTRICTION;

EID: 84982181813     PISSN: 03008177     EISSN: 15734919     Source Type: Journal    
DOI: 10.1007/s11010-016-2784-0     Document Type: Article

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