Progressive postnatal decline in leptin sensitivity of arcuate hypothalamic neurons in the Magel2-null mouse model of Prader–Willi syndrome

Human Molecular Genetics

Volumn 24, Issue 15, 2015, Pages 4276-4283

  Pravdivyi, Igor ^a   Ballanyi, Klaus ^b   Colmers, William F ^a,b   Wevrick, Rachel ^a,b  

^a UNIVERSITY OF ALBERTA   (Canada)

^b UNIVERSITY OF ALBERTA   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

ALPHA INTERMEDIN; CALCIUM; LEPTIN; PROOPIOMELANOCORTIN; MAGEL2 PROTEIN, MOUSE; PROTEIN; TUMOR ANTIGEN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; APPETITE; ARTICLE; BODY WEIGHT GAIN; CLINICAL ARTICLE; DEPOLARIZATION; DEVELOPMENTAL DELAY; FOOD INTAKE; GENETIC MODEL; HYPERPHAGIA; HYPOTHALAMIC PARAVENTRICULAR NUCLEUS; HYPOTHALAMIC SLICE; IMMUNOFLUORESCENCE MICROSCOPY; IMMUNOREACTIVITY; IN VITRO STUDY; LIPID DIET; MOUSE; NERVE CELL; NONHUMAN; OBESITY; POSTNATAL DEVELOPMENT; PRADER WILLI SYNDROME; WILD TYPE; ANIMAL; ARCUATE NUCLEUS; DISEASE MODEL; GENETICS; GROWTH, DEVELOPMENT AND AGING; HUMAN; HYPOTHALAMUS; METABOLISM; PATHOLOGY; WEIGHT GAIN;

ANIMALS; ANTIGENS, NEOPLASM; ARCUATE NUCLEUS OF HYPOTHALAMUS; DISEASE MODELS, ANIMAL; HUMANS; HYPOTHALAMUS; LEPTIN; MICE; NEURONS; PRADER-WILLI SYNDROME; PRO-OPIOMELANOCORTIN; PROTEINS; WEIGHT GAIN;

EID: 84973432165     PISSN: 09646906     EISSN: 14602083     Source Type: Journal    
DOI: 10.1093/hmg/ddv159     Document Type: Article

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