-
1
-
-
84877102781
-
Beyond pattern recognition: NOD-like receptors in dendritic cells
-
Krishnaswamy JK, Chu T, Eisenbarth SC. Beyond pattern recognition: NOD-like receptors in dendritic cells. Trends Immunol 2013; 34:224-233.
-
(2013)
Trends Immunol
, vol.34
, pp. 224-233
-
-
Krishnaswamy, J.K.1
Chu, T.2
Eisenbarth, S.C.3
-
2
-
-
79959634861
-
Pattern recognition receptors and the innate immune response to viral infection
-
Thompson MR, Kaminski JJ, Kurt-Jones EA, Fitzgerald KA. Pattern recognition receptors and the innate immune response to viral infection. Viruses 2011; 3:920-940.
-
(2011)
Viruses
, vol.3
, pp. 920-940
-
-
Thompson, M.R.1
Kaminski, J.J.2
Kurt-Jones, E.A.3
Fitzgerald, K.A.4
-
3
-
-
24144461689
-
Identification and characterization of MAVS, a mitochondrial antiviral signaling protein that activates NF-kappaB and IRF 3
-
Seth RB, Sun L, Ea CK, Chen ZJ. Identification and characterization of MAVS, a mitochondrial antiviral signaling protein that activates NF-kappaB and IRF 3. Cell 2005; 122:669-682.
-
(2005)
Cell
, vol.122
, pp. 669-682
-
-
Seth, R.B.1
Sun, L.2
Ea, C.K.3
Chen, Z.J.4
-
4
-
-
84906794202
-
Persistent inflammation in HIV infection: Established concepts, new perspectives
-
Nasi M, Pinti M, Mussini C, Cossarizza A. Persistent inflammation in HIV infection: established concepts, new perspectives. Immunol Lett 2014; 161:184-188.
-
(2014)
Immunol Lett
, vol.161
, pp. 184-188
-
-
Nasi, M.1
Pinti, M.2
Mussini, C.3
Cossarizza, A.4
-
5
-
-
77950952763
-
Cytotoxic granule release dominates gag-specific CD4R T-cell response in different phases of HIV infection
-
Nemes E, Bertoncelli L, Lugli E, Pinti M, Nasi M, Manzini L, et al. Cytotoxic granule release dominates gag-specific CD4R T-cell response in different phases of HIV infection. AIDS 2010; 24:947-957.
-
(2010)
AIDS
, vol.24
, pp. 947-957
-
-
Nemes, E.1
Bertoncelli, L.2
Lugli, E.3
Pinti, M.4
Nasi, M.5
Manzini, L.6
-
6
-
-
84905376910
-
HIV-1 infection induces interleukin-1beta production via TLR8 protein-dependent and NLRP3 inflammasome mechanisms in human mono-cytes
-
Guo H, Gao J, Taxman DJ, Ting JP, Su L. HIV-1 infection induces interleukin-1beta production via TLR8 protein-dependent and NLRP3 inflammasome mechanisms in human mono-cytes. J Biol Chem 2014; 289:21716-21726.
-
(2014)
J Biol Chem
, vol.289
, pp. 21716-21726
-
-
Guo, H.1
Gao, J.2
Taxman, D.J.3
Ting, J.P.4
Su, L.5
-
7
-
-
84901703352
-
HIV and HCV activate the inflammasome in monocytes and macrophages via endosomal Toll-like receptors without induction of type 1 interferon
-
Chattergoon MA, Latanich R, Quinn J, Winter ME, Buckheit RW, Blankson JN, et al. HIV and HCV activate the inflammasome in monocytes and macrophages via endosomal Toll-like receptors without induction of type 1 interferon. PLoS Pathog 2014; 10:e1004082.
-
(2014)
PLoS Pathog
, vol.10
, pp. e1004082
-
-
Ma, C.1
Latanich, R.2
Quinn, J.3
Winter, M.E.4
Buckheit, R.W.5
Blankson, J.N.6
-
8
-
-
38749097018
-
NLRX1 is a regulator of mitochondrial antiviral immunity
-
Moore CB, Bergstralh DT, Duncan JA, Lei Y, Morrison TE, Zimmermann AG, et al. NLRX1 is a regulator of mitochondrial antiviral immunity. Nature 2008; 451:573-577.
-
(2008)
Nature
, vol.451
, pp. 573-577
-
-
Moore, C.B.1
Bergstralh, D.T.2
Duncan, J.A.3
Lei, Y.4
Morrison, T.E.5
Zimmermann, A.G.6
-
9
-
-
40249111682
-
NLRX1 is a mitochondrial NOD-like receptor that amplifies NF-(B and JNK pathwaysby inducing reactive oxygen species production
-
Tattoli I, Carneiro LA, Jéhanno M, Magalhaes JG, Shu Y, Philpott DJ, et al. NLRX1 is a mitochondrial NOD-like receptor that amplifies NF-(B and JNK pathwaysby inducing reactive oxygen species production. EMBO Rep 2008; 9:293-300.
-
(2008)
EMBO Rep
, vol.9
, pp. 293-300
-
-
Tattoli, I.1
Carneiro, L.A.2
Jéhanno, M.3
Magalhaes, J.G.4
Shu, Y.5
Philpott, D.J.6
-
10
-
-
84904177856
-
The mitochondrial protein NLRX1 controls the balance between extrinsic and intrinsic apoptosis
-
Soares F, Tattoli I, Rahman MA, Robertson SJ, Belcheva A, Liu D, et al. The mitochondrial protein NLRX1 controls the balance between extrinsic and intrinsic apoptosis. J Biol Chem 2014; 289:19317-19330.
-
(2014)
J Biol Chem
, vol.289
, pp. 19317-19330
-
-
Soares, F.1
Tattoli, I.2
Ma, R.3
Robertson, S.J.4
Belcheva, A.5
Liu, D.6
-
11
-
-
84901002026
-
NLRX1 prevents mitochondrial induced apop-tosis and enhances macrophage antiviral immunity by interacting with influenza virus PB1-F2 protein
-
Jaworskaa J, Coulombea F, Downeya J, Tzelepisa F, Shalabya K, Tattoli I, et al. NLRX1 prevents mitochondrial induced apop-tosis and enhances macrophage antiviral immunity by interacting with influenza virus PB1-F2 protein. Proc Natl Acad Sci USA 2014; 111:2110-2119.
-
(2014)
Proc Natl Acad Sci USA
, vol.111
, pp. 2110-2119
-
-
Jaworskaa, J.1
Coulombea, F.2
Downeya, J.3
Tzelepisa, F.4
Shalabya, K.5
Tattoli, I.6
-
12
-
-
84863005844
-
The mitochondrial proteins NLRX1 and TUFM form a complex that regulates type i interferon and autophagy
-
Lei Y, Wen H, Yu Y, Taxman DJ, Zhang L, Widman DG, et al. The mitochondrial proteins NLRX1 and TUFM form a complex that regulates type I interferon and autophagy. Immunity 2012; 36:933-946.
-
(2012)
Immunity
, vol.36
, pp. 933-946
-
-
Lei, Y.1
Wen, H.2
Yu, Y.3
Taxman, D.J.4
Zhang, L.5
Widman, D.G.6
-
13
-
-
84911948494
-
Nucleoside reverse transcriptase inhibitors possess intrinsic anti-inflammatory activity
-
Fowler BJ, Gelfand BD, Kim Y, Kerur N, Tarallo V, Hirano Y, et al. Nucleoside reverse transcriptase inhibitors possess intrinsic anti-inflammatory activity. Science 2014; 346:1000-1003.
-
(2014)
Science
, vol.346
, pp. 1000-1003
-
-
Fowler, B.J.1
Gelfand, B.D.2
Kim, Y.3
Kerur, N.4
Tarallo, V.5
Hirano, Y.6
-
14
-
-
84922061007
-
Successful treatment of HIV-1 infection increases the expression of a novel, short transcript for IL-18 receptor alpha chain
-
Nasi M, Alboni S, Pinti M, Tascedda F, Benatti C, Benatti S, et al. Successful treatment of HIV-1 infection increases the expression of a novel, short transcript for IL-18 receptor alpha chain. J Acquir Immune Defic Syndr 2014; 67:254-257.
-
(2014)
J Acquir Immune Defic Syndr
, vol.67
, pp. 254-257
-
-
Nasi, M.1
Alboni, S.2
Pinti, M.3
Tascedda, F.4
Benatti, C.5
Benatti, S.6
-
15
-
-
62649092113
-
Mechanism of Bcl-2 and Bcl-X(L) inhibition of NLRP1 inflammasome: Loop domain-dependent suppression of ATP binding and oligomerization
-
Faustin B, Chen Y, Zhai D, Le Negrate G, Lartigue L, Satterthwait A, et al. Mechanism of Bcl-2 and Bcl-X(L) inhibition of NLRP1 inflammasome: loop domain-dependent suppression of ATP binding and oligomerization. Proc Natl Acad Sci USA 2009; 106:3935-3940.
-
(2009)
Proc Natl Acad Sci USA
, vol.106
, pp. 3935-3940
-
-
Faustin, B.1
Chen, Y.2
Zhai, D.3
Le Negrate, G.4
Lartigue, L.5
Satterthwait, A.6
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