Altered Striatal Synaptic Function and Abnormal Behaviour in Shank3 Exon4-9 Deletion Mouse Model of Autism

Autism Research

Volumn 9, Issue 3, 2016, Pages 350-375

  Jaramillo, Thomas C ^a   Speed, Haley E ^a   Xuan, Zhong ^a   Reimers, Jeremy M ^a   Liu, Shunan ^a   Powell, Craig M ^a  

^a UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

Author keywords

Autism spectrum disorder; Grooming; Mouse model; Phelan McDermid syndrome; Shank3

Indexed keywords

ALPHA AMINO 3 HYDROXY 5 METHYL 4 ISOXAZOLEPROPIONIC ACID; AMPA RECEPTOR; ISOPROTEIN; N METHYL DEXTRO ASPARTIC ACID; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; PROTEIN HOMER 1B; PROTEIN HOMER 1C; RECEPTOR SUBUNIT; SCAFFOLD PROTEIN; SHANK3; UNCLASSIFIED DRUG; NERVE PROTEIN; SHANK3 PROTEIN, MOUSE;

ABNORMAL BEHAVIOR; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; AUTISM; BRAIN FUNCTION; COGNITION; CONTROLLED STUDY; CORPUS STRIATUM; EXON; FEMALE; GENE DELETION; GENE MUTATION; GROOMING; HIPPOCAMPUS; KNOCKOUT MOUSE; LANGUAGE DELAY; LANGUAGE DISABILITY; LEARNING; LONG TERM POTENTIATION; MALE; MEMORY; MOUSE; MOUSE MODEL; NONHUMAN; OBSESSIVE COMPULSIVE DISORDER; PATHOPHYSIOLOGY; POSTSYNAPTIC DENSITY; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN LOCALIZATION; SOCIAL INTERACTION; SPEECH DELAY; SPEECH DISORDER; SYNAPSE; SYNAPTIC TRANSMISSION; SYNAPTOSOME; VOCALIZATION; ANIMAL; ANIMAL BEHAVIOR; DISEASE MODEL; GENETICS; PHYSIOLOGY;

ANIMALS; AUTISTIC DISORDER; BEHAVIOR, ANIMAL; CORPUS STRIATUM; DISEASE MODELS, ANIMAL; EXONS; FEMALE; MALE; MICE; NERVE TISSUE PROTEINS; SEQUENCE DELETION; SYNAPSES; SYNAPTIC TRANSMISSION;

EID: 84961240619     PISSN: 19393792     EISSN: 19393806     Source Type: Journal    
DOI: 10.1002/aur.1529     Document Type: Article

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