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Volumn 318, Issue 5847, 2007, Pages 71-76
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A neuroligin-3 mutation implicated in autism increases inhibitory synaptic transmission in mice
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Author keywords
[No Author keywords available]
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Indexed keywords
NEUROLIGIN;
NEUROLIGIN 3;
UNCLASSIFIED DRUG;
ECOLOGICAL MODELING;
GENE EXPRESSION;
LEARNING;
MUTATION;
RODENT;
AMINO ACID SUBSTITUTION;
ANIMAL BEHAVIOR;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ARTICLE;
AUTISM;
BEHAVIOR CHANGE;
BEHAVIORAL SCIENCE;
CONTROLLED STUDY;
GENE DELETION;
GENE MUTATION;
INHIBITORY POSTSYNAPTIC POTENTIAL;
MISSENSE MUTATION;
MOUSE;
MUTATIONAL ANALYSIS;
NERVE CELL EXCITABILITY;
NONHUMAN;
POSTSYNAPTIC POTENTIAL;
PRIORITY JOURNAL;
SYNAPTIC TRANSMISSION;
AMINO ACID SUBSTITUTION;
ANIMALS;
AUTISTIC DISORDER;
BRAIN;
DISEASE MODELS, ANIMAL;
FEMALE;
GENE TARGETING;
HIPPOCAMPUS;
HUMANS;
MALE;
MAZE LEARNING;
MEMBRANE PROTEINS;
MEMORY;
MICE;
MICE, KNOCKOUT;
MUTATION;
NERVE TISSUE PROTEINS;
SOCIAL BEHAVIOR;
SOMATOSENSORY CORTEX;
SYNAPSES;
SYNAPTIC TRANSMISSION;
VESICULAR GLUTAMATE TRANSPORT PROTEIN 1;
VESICULAR INHIBITORY AMINO ACID TRANSPORT PROTEINS;
MUS;
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EID: 35148858044
PISSN: 00368075
EISSN: 10959203
Source Type: Journal
DOI: 10.1126/science.1146221 Document Type: Article |
Times cited : (792)
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References (38)
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