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Volumn 249, Issue 4, 2016, Pages 437-447

Anti-tumor Necrosis Factor Alpha (Infliximab) Attenuates Apoptosis, Oxidative Stress, and Calcium Ion Entry Through Modulation of Cation Channels in Neutrophils of Patients with Ankylosing Spondylitis

Author keywords

Ankylosing spondylitis; Apoptosis; Calcium signaling; Infliximab; Neutrophil; Oxidative stress

Indexed keywords

2 AMINOETHOXYDIPHENYLBORANE; C REACTIVE PROTEIN; CALCIUM ION; CASPASE 3; CASPASE 9; DILTIAZEM; FORMYLMETHIONYLLEUCYLPHENYLALANINE; GLUTATHIONE; GLUTATHIONE PEROXIDASE; INFLIXIMAB; MALONALDEHYDE; REACTIVE OXYGEN METABOLITE; THIOBARBITURIC ACID REACTIVE SUBSTANCE; TRANSIENT RECEPTOR POTENTIAL CHANNEL M2; VERAPAMIL; VOLTAGE GATED CALCIUM CHANNEL; ANTIRHEUMATIC AGENT; BIOLOGICAL MARKER; CALCIUM; CALCIUM CHANNEL;

EID: 84960120477     PISSN: 00222631     EISSN: 14321424     Source Type: Journal    
DOI: 10.1007/s00232-016-9884-3     Document Type: Article
Times cited : (13)

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