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Volumn 49, Issue 3, 2015, Pages 338-346

N-acetyl cysteine reduces oxidative toxicity, apoptosis, and calcium entry through TRPV1 channels in the neutrophils of patients with polycystic ovary syndrome

Author keywords

Antioxidant; Inflammation; N acetyl cysteine; neutrophil; Polycystic ovary syndrome; TRPV1 channel

Indexed keywords

ACETYLCYSTEINE; ALPHA TOCOPHEROL; BETA CAROTENE; CASPASE 3; CASPASE 9; FOLLITROPIN; GLUCOSE; GLUTATHIONE; GLUTATHIONE PEROXIDASE; HOMOCYSTEINE; INSULIN; INTERLEUKIN 1BETA; LUTEINIZING HORMONE; REACTIVE OXYGEN METABOLITE; RETINOL; TESTOSTERONE; VANILLOID RECEPTOR 1; ANTIOXIDANT; CALCIUM; CYTOKINE; TRANSIENT RECEPTOR POTENTIAL CHANNEL M; TRPM2 PROTEIN, HUMAN; TRPV1 PROTEIN, HUMAN; VANILLOID RECEPTOR;

EID: 84925782428     PISSN: 10715762     EISSN: 10292470     Source Type: Journal    
DOI: 10.3109/10715762.2015.1006214     Document Type: Article
Times cited : (35)

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