Increased susceptibility of H-Ras G12V-transformed human urothelial cells to the genotoxic effects of sodium arsenite

Archives of Toxicology

Volumn 89, Issue 11, 2015, Pages 1971-1979

  Liao, Yi Chun ^a   Chen, Yi Fan ^b   Lee, Te Chang ^a,b  

^a NATIONAL YANG MING UNIVERSITY   (Taiwan)

^b INSTITUTE OF BIOMEDICAL SCIENCES   (Taiwan)

Author keywords

Arsenic; Catalase; Genotoxicity; H Ras oncogene; Reactive oxygen species; Stress signaling

Indexed keywords

ACETYLCYSTEINE; ARSENITE SODIUM; CATALASE; GLUTATHIONE; MITOGEN ACTIVATED PROTEIN KINASE; REACTIVE OXYGEN METABOLITE; STRESS ACTIVATED PROTEIN KINASE; SUPEROXIDE DISMUTASE; ARSENOUS ACID DERIVATIVE; MUTAGENIC AGENT; SODIUM DERIVATIVE;

ANCHORAGE INDEPENDENT GROWTH; ARTICLE; CELL TRANSFORMATION; CONTROLLED STUDY; CYTOTOXICITY; DNA DAMAGE; ENZYME ACTIVITY; GENE MUTATION; GENETIC DAMAGE; GENETIC SUSCEPTIBILITY; GENOTOXICITY; HUMAN; HUMAN CELL; MICRONUCLEUS; ONCOGENE H RAS; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; TRANSITIONAL CELL CARCINOMA; CELL DEATH; CELL LINE; CYTOLOGY; DRUG EFFECTS; EPITHELIUM CELL; GENETICS; METABOLISM; ONCOGENE RAS; PATHOLOGY; UROTHELIUM;

ACETYLCYSTEINE; ARSENITES; CELL DEATH; CELL LINE; DNA DAMAGE; EPITHELIAL CELLS; GENES, RAS; GLUTATHIONE; HUMANS; MAP KINASE SIGNALING SYSTEM; MUTAGENS; REACTIVE OXYGEN SPECIES; SODIUM COMPOUNDS; SUPEROXIDE DISMUTASE; UROTHELIUM;

EID: 84947550702     PISSN: 03405761     EISSN: 14320738     Source Type: Journal    
DOI: 10.1007/s00204-014-1344-1     Document Type: Article

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