XPC silencing sensitizes glioma cells to arsenic trioxide via increased oxidative damage

Toxicological Sciences

Volumn 116, Issue 1, 2010, Pages 183-193

  Liu, Shin Yi ^a   Wen, Ching Ya ^a   Lee, Yi Jang ^a   Lee, Te Chang ^a,b  

^a NATIONAL YANG MING UNIVERSITY   (Taiwan)

^b INSTITUTE OF BIOMEDICAL SCIENCES   (Taiwan)

Author keywords

Antioxidant activity; Arsenic trioxide; Catalase; Oxidative damage; RNA interference; XPC

Indexed keywords

8 HYDROXYDEOXYGUANOSINE; ARSENIC TRIOXIDE; CATALASE; DNA; GLUTATHIONE; MANGANESE SUPEROXIDE DISMUTASE; PEROXIREDOXIN; REACTIVE OXYGEN METABOLITE; SMALL INTERFERING RNA; SUPEROXIDE; TRANSCRIPTION FACTOR AP 1; XERODERMA PIGMENTOSUM GROUP C PROTEIN; DNA BINDING PROTEIN; ORGANOARSENIC DERIVATIVE; OXIDE; XPC PROTEIN, HUMAN;

ARTICLE; AUTOPHAGY; CANCER CELL CULTURE; CELL DEATH; CONTROLLED STUDY; CYTOTOXICITY; DNA REPAIR; DNA STRAND BREAKAGE; DRUG POTENTIATION; DRUG SENSITIZATION; ENZYME ACTIVITY; GENE SILENCING; GLIOMA; GLIOMA CELL; HUMAN; HUMAN CELL; OXIDATION REDUCTION STATE; OXIDATIVE STRESS; PHARMACOGENETICS; REPORTER GENE; SENESCENCE; BRAIN TUMOR; CELL AGING; FLUORESCENT ANTIBODY TECHNIQUE; GENETICS; METABOLISM; NUCLEOTIDE SEQUENCE; PATHOLOGY; TOXICITY; TUMOR CELL LINE;

ARSENICALS; AUTOPHAGY; BASE SEQUENCE; BRAIN NEOPLASMS; CELL AGING; CELL LINE, TUMOR; DNA-BINDING PROTEINS; FLUORESCENT ANTIBODY TECHNIQUE; GENE SILENCING; GLIOMA; HUMANS; OXIDATIVE STRESS; OXIDES; RNA, SMALL INTERFERING;

EID: 77954350444     PISSN: 10966080     EISSN: 10960929     Source Type: Journal    
DOI: 10.1093/toxsci/kfq113     Document Type: Article

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