Noise-induced plasticity of KCNQ2/3 and HCN channels underlies vulnerability and resilience to tinnitus

eLife

Volumn 4, Issue AUGUST2015, 2015, Pages

  Li, Shuang ^a   Kalappa, Bopanna I ^a   Tzounopoulo, Thanos ^a  

^a UNIVERSITY OF PITTSBURGH SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

HYPERPOLARIZATION ACTIVATED CYCLIC NUCLEOTIDE GATED CHANNEL; POTASSIUM CHANNEL KCNQ2; KCNQ2 PROTEIN, MOUSE; KCNQ3 PROTEIN, MOUSE; NERVE PROTEIN; POTASSIUM CHANNEL KCNQ3;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BIOPHYSICS; BRAIN DISEASE; CONDUCTANCE; CONTROLLED STUDY; DISEASE PREDISPOSITION; DISEASE RESISTANCE; DORSAL COCHLEAR NUCLEUS; ELECTRICAL SYNAPSE; ELECTROPHYSIOLOGICAL PROCEDURES; FEMALE; FIRING RATE; HYPERPOLARIZATION; MALE; MOUSE; NERVE CELL PLASTICITY; NOISE; NONHUMAN; POTASSIUM CURRENT; RECEPTOR INTRINSIC ACTIVITY; SPINDLE CELL; TINNITUS; ANIMAL; METABOLISM; PATHOPHYSIOLOGY;

ANIMALS; HYPERPOLARIZATION-ACTIVATED CYCLIC NUCLEOTIDE-GATED CHANNELS; KCNQ2 POTASSIUM CHANNEL; KCNQ3 POTASSIUM CHANNEL; MICE; NERVE TISSUE PROTEINS; NOISE; TINNITUS;

EID: 84943743248     PISSN: None     EISSN: 2050084X     Source Type: Journal    
DOI: 10.7554/eLife.0724     Document Type: Article

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