SETDB1, HP1 and SUV39 promote repositioning of 53BP1 to extend resection during homologous recombination in G2 cells

Nucleic Acids Research

Volumn 43, Issue 16, 2015, Pages 7931-7944

  Alagoz, Meryem ^a   Katsuki, Yoko ^a,e   Ogiwara, Hideaki ^b   Ogi, Tomoo ^c   Shibata, Atsushi ^a,f   Kakarougkas, Andreas ^a,d   Jeggo, Penny ^a  

^a UNIVERSITY OF SUSSEX   (United Kingdom)

^b NATIONAL CANCER CENTER   (Japan)

^c NAGASAKI UNIVERSITY   (Japan)

^d The American University in Cairo   (Egypt)

^e KYOTO UNIVERSITY   (Japan)

^f GUNMA UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

BRCA1 PROTEIN; DNA; HETEROCHROMATIN PROTEIN 1; NUCLEAR PROTEIN; PHOSPHOPROTEIN; PROTEIN 53BP1; PROTEIN SETDB1; PROTEIN SUV39; RAD51 PROTEIN; REPLICATION FACTOR A; SMALL INTERFERING RNA; UNCLASSIFIED DRUG; HETEROCHROMATIN-SPECIFIC NONHISTONE CHROMOSOMAL PROTEIN HP-1; HISTONE LYSINE METHYLTRANSFERASE; METHYLTRANSFERASE; NONHISTONE PROTEIN; PROTEIN METHYLTRANSFERASE; REPRESSOR PROTEIN; SETDB1 PROTEIN, HUMAN; SIGNAL PEPTIDE; SUV39H1 PROTEIN, HUMAN; SUV39H2 PROTEIN, HUMAN; TP53BP1 PROTEIN, HUMAN;

ARTICLE; CELL CULTURE; CELL CYCLE G2 PHASE; CONTROLLED STUDY; DNA DAMAGE; FIBROBLAST; HISTONE METHYLATION; HOMOLOGOUS RECOMBINATION; HUMAN; HUMAN CELL; IONIZING RADIATION; PHENOTYPE; PRIORITY JOURNAL; PROTEIN DEPLETION; PROTEIN LOCALIZATION; SISTER CHROMATID; ANTAGONISTS AND INHIBITORS; CHROMATID; DNA END JOINING REPAIR; METABOLISM; PHYSIOLOGY; RECOMBINATION REPAIR;

BRCA1 PROTEIN; CELLS, CULTURED; CHROMATIDS; CHROMOSOMAL PROTEINS, NON-HISTONE; DNA DAMAGE; DNA END-JOINING REPAIR; G2 PHASE; HISTONE-LYSINE N-METHYLTRANSFERASE; HUMANS; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; METHYLTRANSFERASES; PROTEIN METHYLTRANSFERASES; RECOMBINATIONAL DNA REPAIR; REPRESSOR PROTEINS;

EID: 84942279276     PISSN: 03051048     EISSN: 13624962     Source Type: Journal    
DOI: 10.1093/nar/gkv722     Document Type: Article

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