P62 deficiency enhances α-synuclein pathology in mice

Brain Pathology

Volumn 25, Issue 5, 2015, Pages 552-564

  Tanji, Kunikazu ^a   Odagiri, Saori ^a   Miki, Yasuo ^a   Maruyama, Atsushi ^a   Nikaido, Yoshikazu ^b   Mimura, Junsei ^a   Mori, Fumiaki ^a   Warabi, Eiji ^b   Yanagawa, Toru ^b   Ueno, Shinya ^b   Itoh, Ken ^a   Wakabayashi, Koichi ^a  

^a HIROSAKI UNIVERSITY   (Japan)

^b UNIVERSITY OF TSUKUBA   (Japan)

Author keywords

Lewy body disease; p62 Sequestsome 1 SQSTM1; Parkinson's disease; proteolysis; stress; synuclein

Indexed keywords

ALPHA SYNUCLEIN; BRCA1 PROTEIN; MESSENGER RNA; NBR1 PROTEIN; PROTEIN P62; UNCLASSIFIED DRUG; HEAT SHOCK PROTEIN; NBR1 PROTEIN, MOUSE; PROTEIN; SIGNAL TRANSDUCING ADAPTOR PROTEIN; SQSTM1 PROTEIN, MOUSE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL INCLUSION; CONTROLLED STUDY; DEGENERATIVE DISEASE; DIFFUSE LEWY BODY DISEASE; GENE EXPRESSION; GENE LOSS; IMMUNOREACTIVITY; MOLECULE; MOUSE; NEUROPATHOLOGY; NONHUMAN; P62 DEFICIENCY; PATHOGENESIS; PROTEIN AGGREGATION; PROTEIN DEFICIENCY; PROTEIN DEGRADATION; PROTEIN STRUCTURE; STAINING; TUMOR SUPPRESSOR GENE; ANIMAL; BRAIN; DEFICIENCY; GENETICS; HUMAN; METABOLISM; MOTOR ACTIVITY; PATHOLOGY; PHYSIOLOGICAL STRESS; TRANSGENIC MOUSE;

ADAPTOR PROTEINS, SIGNAL TRANSDUCING; ALPHA-SYNUCLEIN; ANIMALS; BRAIN; HEAT-SHOCK PROTEINS; HUMANS; INCLUSION BODIES; LEWY BODY DISEASE; MICE; MICE, TRANSGENIC; MOTOR ACTIVITY; PROTEINS; PROTEOLYSIS; STRESS, PHYSIOLOGICAL;

EID: 84939442884     PISSN: 10156305     EISSN: 17503639     Source Type: Journal    
DOI: 10.1111/bpa.12214     Document Type: Article

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