Ischemic postconditioning provides cardioprotective and antiapoptotic effects against ischemia-reperfusion injury through iNOS inhibition in hyperthyroid rats

Gene

Volumn 570, Issue 2, 2015, Pages 185-190

  Zaman, Jalal ^a,c   Jeddi, Sajad ^a,c   Daneshpour, Maryam Sadat ^b   Zarkesh, Maryam ^b   Daneshian, Zahra ^b   Ghasemi, Asghar ^a,c  

^a SHAHID BEHESHTI UNIVERSITY OF MEDICAL SCIENCES   (Iran)

^b SHAHID BEHESHTI UNIVERSITY OF MEDICAL SCIENCES   (Iran)

^c SHAHID BEHESHTI UNIVERSITY OF MEDICAL SCIENCES   (Iran)

Author keywords

Gene expression; Hyperthyroidism; Ischemia; Postconditioning

Indexed keywords

AMINOGUANIDINE; BETA ACTIN; CREATINE KINASE; ENDOTHELIAL NITRIC OXIDE SYNTHASE; INDUCIBLE NITRIC OXIDE SYNTHASE; LACTATE DEHYDROGENASE; LIOTHYRONINE; PROTEIN BAX; PROTEIN BCL 2; THYROID HORMONE; THYROTROPIN; THYROXINE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; COMORBIDITY; CONTROLLED STUDY; HEART INFARCTION SIZE; HEART LEFT VENTRICLE; HEART MUSCLE ISCHEMIA; HEART MUSCLE REPERFUSION; HEART PROTECTION; HEART WEIGHT; HYPERTHYROIDISM; ISCHEMIC POSTCONDITIONING; MALE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; REPERFUSION INJURY; WEIGHT CHANGE; ANIMAL; ANTAGONISTS AND INHIBITORS; APOPTOSIS; ENZYMOLOGY; PATHOLOGY; WISTAR RAT;

ANIMALIA; RATTUS;

ANIMALS; APOPTOSIS; HYPERTHYROIDISM; ISCHEMIC POSTCONDITIONING; MALE; NITRIC OXIDE SYNTHASE TYPE II; RATS; RATS, WISTAR; REPERFUSION INJURY;

EID: 84938954752     PISSN: 03781119     EISSN: 18790038     Source Type: Journal    
DOI: 10.1016/j.gene.2015.06.011     Document Type: Article

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