-
1
-
-
0022970945
-
Preconditioning with ischemia: A delay of lethal cell injury in ischemic myocardium
-
Murry CE, Jennings RB, Reimer KA. Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium. Circulation. 1986;74:1124-1136.
-
(1986)
Circulation
, vol.74
, pp. 1124-1136
-
-
Murry, C.E.1
Jennings, R.B.2
Reimer, K.A.3
-
2
-
-
0037623908
-
Inhibition of myocardial injury by ischemic post-conditioning during reperfusion: Comparison with ischemic preconditioning
-
Zhao ZQ, Corvera JS, Halkos ME, Kerendi F, Wang NP, Guyton RA, Vinten-Johansen J. Inhibition of myocardial injury by ischemic post-conditioning during reperfusion: comparison with ischemic preconditioning. Am J Physiol Heart Circ Physiol. 2003;285:H579-H588.
-
(2003)
Am J Physiol Heart Circ Physiol
, vol.285
-
-
Zhao, Z.Q.1
Corvera, J.S.2
Halkos, M.E.3
Kerendi, F.4
Wang, N.P.5
Guyton, R.A.6
Vinten-Johansen, J.7
-
3
-
-
1542298950
-
Postconditioning attenuates myocardial ischemia-reperfusion injury by inhibiting events in the early minutes of reperfusion
-
Kin H, Zhao ZQ, Sun HY, Wang NP, Corvera JS, Halkos ME, Kerendi F, Guyton RA, Vinten-Johansen J. Postconditioning attenuates myocardial ischemia-reperfusion injury by inhibiting events in the early minutes of reperfusion. Cardiovasc Res. 2004;62:74-85.
-
(2004)
Cardiovasc Res
, vol.62
, pp. 74-85
-
-
Kin, H.1
Zhao, Z.Q.2
Sun, H.Y.3
Wang, N.P.4
Corvera, J.S.5
Halkos, M.E.6
Kerendi, F.7
Guyton, R.A.8
Vinten-Johansen, J.9
-
4
-
-
4043076539
-
Hypoxic post-conditioning reduces cardiomyocyte loss by inhibiting reactive oxygen species-triggered mitochondrial calcium overload
-
Zhao ZQ, Sun HY, Wang NP, Kerendi F, Guyton RA, Vinten-Johansen J. Hypoxic post-conditioning reduces cardiomyocyte loss by inhibiting reactive oxygen species-triggered mitochondrial calcium overload. Circulation. 2003;108(suppl IV):174.
-
(2003)
Circulation
, vol.108
, Issue.SUPPL. IV
, pp. 174
-
-
Zhao, Z.Q.1
Sun, H.Y.2
Wang, N.P.3
Kerendi, F.4
Guyton, R.A.5
Vinten-Johansen, J.6
-
5
-
-
1142309743
-
New directions for protecting the heart against ischemia-reperfusion injury: Targeting the reperfusion injury salvage kinase (RISK)-pathway
-
Hausenloy DJ, Yellon DM. New directions for protecting the heart against ischemia-reperfusion injury: targeting the reperfusion injury salvage kinase (RISK)-pathway. Cardiovasc Res. 2004;61:448-460.
-
(2004)
Cardiovasc Res
, vol.61
, pp. 448-460
-
-
Hausenloy, D.J.1
Yellon, D.M.2
-
6
-
-
0345490687
-
Inhibiting mitochondrial permeability transition pore opening at reperfusion protects against ischaemia-reperfusion injury
-
Hausenloy DJ, Duchen MR, Yellon DM. Inhibiting mitochondrial permeability transition pore opening at reperfusion protects against ischaemia-reperfusion injury. Cardiovasc Res. 2003;60:617-625.
-
(2003)
Cardiovasc Res
, vol.60
, pp. 617-625
-
-
Hausenloy, D.J.1
Duchen, M.R.2
Yellon, D.M.3
-
7
-
-
0036623989
-
PI3 kinase and not p42/p44 appears to be implicated in the protection conferred by ischemic preconditioning
-
Mocanu MM, Bell RM, Yellon DM. PI3 kinase and not p42/p44 appears to be implicated in the protection conferred by ischemic preconditioning. J Mol Cell Cardiol. 2002;34:661-668.
-
(2002)
J Mol Cell Cardiol
, vol.34
, pp. 661-668
-
-
Mocanu, M.M.1
Bell, R.M.2
Yellon, D.M.3
-
8
-
-
4043170109
-
Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by activation of ERK and production of nitric oxide
-
Yang X, Downey JM, Cohen MV. Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by activation of ERK and production of nitric oxide. Circulation. 2003;108(suppl IV):158.
-
(2003)
Circulation
, vol.108
, Issue.SUPPL. IV
, pp. 158
-
-
Yang, X.1
Downey, J.M.2
Cohen, M.V.3
-
9
-
-
0028968606
-
Mitochondrial non-specific pores remain closed during cardiac ischemia, but open upon reperfusion
-
Griffiths EJ, Halestrap AP. Mitochondrial non-specific pores remain closed during cardiac ischemia, but open upon reperfusion. Biochem J. 1995;307:93-98.
-
(1995)
Biochem J
, vol.307
, pp. 93-98
-
-
Griffiths, E.J.1
Halestrap, A.P.2
-
10
-
-
0036023641
-
Inhibiting mitochondrial permeability transition pore opening: A new paradigm for myocardial preconditioning?
-
Hausenloy DJ, Maddock HL, Baxter GF, Yellon DM. Inhibiting mitochondrial permeability transition pore opening: a new paradigm for myocardial preconditioning? Cardiovasc Res. 2002;55:534-543.
-
(2002)
Cardiovasc Res
, vol.55
, pp. 534-543
-
-
Hausenloy, D.J.1
Maddock, H.L.2
Baxter, G.F.3
Yellon, D.M.4
-
11
-
-
0037134431
-
The phosphatidylinositol 3-kinase (PI3K)-Akt pathway suppresses Bax translocation to mitochondria
-
Tsuruta F, Masuyama N, Gotoh Y. The phosphatidylinositol 3-kinase (PI3K)-Akt pathway suppresses Bax translocation to mitochondria. J Biol Chem. 2002;277:14040-14047.
-
(2002)
J Biol Chem
, vol.277
, pp. 14040-14047
-
-
Tsuruta, F.1
Masuyama, N.2
Gotoh, Y.3
-
12
-
-
0030941354
-
Modulation of the mitochondrial permeability transition by nitric oxide
-
Balakirev MY, Khramtsov VV, Zimmer G. Modulation of the mitochondrial permeability transition by nitric oxide. Eur J Biochem. 1997;246:710-718.
-
(1997)
Eur J Biochem
, vol.246
, pp. 710-718
-
-
Balakirev, M.Y.1
Khramtsov, V.V.2
Zimmer, G.3
-
13
-
-
0030682497
-
Gradual reperfusion reduces infarct size and endothelial injury but augments neutrophil accumulation
-
Sato H, Jordan JE, Zhao ZQ, Sarvotham SS, Vinten-Johansen J. Gradual reperfusion reduces infarct size and endothelial injury but augments neutrophil accumulation. Ann Thorac Surg. 1997;64:1099-1107.
-
(1997)
Ann Thorac Surg
, vol.64
, pp. 1099-1107
-
-
Sato, H.1
Jordan, J.E.2
Zhao, Z.Q.3
Sarvotham, S.S.4
Vinten-Johansen, J.5
|