Cardiac H2S generation is reduced in ageing diabetic mice

Oxidative Medicine and Cellular Longevity

Volumn 2015, Issue , 2015, Pages

  Jin, Sheng ^a,b   Pu, Shi Xin ^a   Hou, Cui Lan ^a   Ma, Fen Fen ^a   Li, Na ^a   Li, Xing Hui ^a   Tan, Bo ^a   Tao, Bei Bei ^a   Wang, Ming Jie ^a   Zhu, Yi Chun ^a  

^a FUDAN UNIVERSITY   (China)

^b HEBEI MEDICAL UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

ENZYMES; FRUCTOSE; HEART;

CARDIAC HYPERTROPHY; DIABETIC CARDIOMYOPATHY; DIABETIC MICE; HEART TISSUES; IN-CONTROL; SYNTHASES; TAP WATER;

MAMMALS;

3 MERCAPTOPYRUVATE SULFURTRANSFERASE; COLLAGEN TYPE 1; COLLAGEN TYPE 3; CYSTATHIONINE GAMMA LYASE; FRUCTOSE; HYDROGEN SULFIDE; PROTEIN BAX; PROTEIN BCL 2; SULFURTRANSFERASE; TAP WATER; UNCLASSIFIED DRUG; 3-MERCAPTOPYRUVATE SULPHURTRANSFERASE; CYSTATHIONINE BETA SYNTHASE; GLUCOSE; REACTIVE OXYGEN METABOLITE; SODIUM SULFIDE; SULFIDE;

AGING; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL VIABILITY; CONTROLLED STUDY; DIABETES MELLITUS; DIABETIC CARDIOMYOPATHY; ECHOCARDIOGRAPHY; ENZYME BLOOD LEVEL; GLUCOSE TOLERANCE; HEART; HEART DISEASE; HEART LEFT VENTRICLE EJECTION FRACTION; HEART MUSCLE FIBROSIS; HEART MUSCLE INJURY; HEART VENTRICLE HYPERTROPHY; IMPAIRED GLUCOSE TOLERANCE; INSULIN RESISTANCE; INSULIN TOLERANCE TEST; MALE; MOUSE; NON INSULIN DEPENDENT DIABETES MELLITUS; NONHUMAN; PROTEIN EXPRESSION; RAT; ANIMAL; BLOOD; C57BL MOUSE; CARDIAC MUSCLE; CARDIAC MUSCLE CELL; CELL CULTURE; CELL SURVIVAL; CYTOLOGY; DISEASE MODEL; DRUG EFFECTS; EXPERIMENTAL DIABETES MELLITUS; METABOLISM; PATHOLOGY; SPRAGUE DAWLEY RAT; URINE;

MUS;

AGING; ANIMALS; CELL SURVIVAL; CELLS, CULTURED; CYSTATHIONINE BETA-SYNTHASE; CYSTATHIONINE GAMMA-LYASE; DIABETES MELLITUS, EXPERIMENTAL; DISEASE MODELS, ANIMAL; GLUCOSE; HEART; HYDROGEN SULFIDE; MALE; MICE; MICE, INBRED C57BL; MYOCARDIUM; MYOCYTES, CARDIAC; RATS; RATS, SPRAGUE-DAWLEY; REACTIVE OXYGEN SPECIES; SULFIDES; SULFURTRANSFERASES;

EID: 84930660214     PISSN: 19420900     EISSN: 19420994     Source Type: Journal    
DOI: 10.1155/2015/758358     Document Type: Article

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