Cellular and deafness mechanisms underlying connexin mutation-induced hearing loss – A common hereditary deafness

Frontiers in Cellular Neuroscience

Volumn 9, Issue MAY, 2015, Pages

  Wingard, Jeffrey C ^a   Zhao, Hong Bo ^a  

^a UNIVERSITY OF KENTUCKY MEDICAL CENTER   (United States)

Author keywords

Active cochlear amplification; Cochlear development; Cochlear supporting cell; Gap junction; Hair cell; Inner ear; Non syndromic hearing loss; Potassium recycling

Indexed keywords

CALCIUM; CONNEXIN 26; CONNEXIN 30; CONNEXIN 31; CONNEXIN 43; GAP JUNCTION PROTEIN; POTASSIUM;

CELL DEGENERATION; COCHLEA; COCHLEA POTENTIAL; DISTORTION PRODUCT OTOACOUSTIC EMISSION; EVOKED BRAIN STEM AUDITORY RESPONSE; GENE MUTATION; HEARING IMPAIRMENT; HUMAN; NONHUMAN; OUTER HAIR CELL; PHENOTYPE; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN LOCALIZATION; REVIEW; SOUND PRESSURE; SPIRAL GANGLION;

EID: 84930631869     PISSN: 16625102     EISSN: None     Source Type: Journal    
DOI: 10.3389/fncel.2015.00202     Document Type: Review

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