Aberrant hemichannel properties of Cx26 mutations causing skin disease and deafness

American Journal of Physiology - Cell Physiology

Volumn 293, Issue 1, 2007, Pages

  Gerido, Dwan A ^a   DeRosa, Adam M ^a   Richard, Gabriele ^b   White, Thomas W ^a  

^a STONY BROOK UNIVERSITY   (United States)

^b GENEDX   (United States)

Author keywords

Connexin

Indexed keywords

CONNEXIN 26;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CALCIUM BLOOD LEVEL; CELL DEATH; CONTROLLED STUDY; CYTOLYSIS; FEMALE; GENETIC DISORDER; HEARING IMPAIRMENT; HOMEOSTASIS; MUTANT; MUTATION; NONHUMAN; OOCYTE; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; SKIN DISEASE; SYNDROME KID; XENOPUS;

ANIMALS; CALCIUM; CELL DEATH; CONNEXINS; DEAFNESS; EXTRACELLULAR FLUID; FEMALE; GAP JUNCTIONS; GENETIC PREDISPOSITION TO DISEASE; GENOTYPE; HOMEOSTASIS; HUMANS; ICHTHYOSIS; ION CHANNEL GATING; ION CHANNELS; KERATITIS; MEMBRANE POTENTIALS; MICROINJECTIONS; MUTATION; OOCYTES; PHENOTYPE; SYNDROME; XENOPUS LAEVIS;

EID: 34250807258     PISSN: 03636143     EISSN: 15221563     Source Type: Journal    
DOI: 10.1152/ajpcell.00626.2006     Document Type: Article

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