-
1
-
-
84893976816
-
Epidemiology and risk factors for invasive candidiasis
-
PID: 24611015
-
Yapar N (2014) Epidemiology and risk factors for invasive candidiasis. Ther Clin Risk Manag 10:95–105
-
(2014)
Ther Clin Risk Manag
, vol.10
, pp. 95-105
-
-
Yapar, N.1
-
2
-
-
84887317220
-
Azole resistance in Aspergillus fumigatus: a growing public health concern
-
COI: 1:CAS:528:DC%2BC3sXhslSmtrfO, PID: 24126719
-
Vermeulen E et al (2013) Azole resistance in Aspergillus fumigatus: a growing public health concern. Curr Opin Infect Dis 26:493–500
-
(2013)
Curr Opin Infect Dis
, vol.26
, pp. 493-500
-
-
Vermeulen, E.1
-
3
-
-
3042521076
-
The distinct morphogenic states of Candida albicans
-
COI: 1:CAS:528:DC%2BD2cXlt1ansbc%3D, PID: 15223059
-
Sudbery P et al (2004) The distinct morphogenic states of Candida albicans. Trends Microbiol 12:317–324
-
(2004)
Trends Microbiol
, vol.12
, pp. 317-324
-
-
Sudbery, P.1
-
4
-
-
37349015349
-
An integrated model of the recognition of Candida albicans by the innate immune system
-
COI: 1:CAS:528:DC%2BD2sXhsVaqt7jN, PID: 18079743
-
Netea MG et al (2008) An integrated model of the recognition of Candida albicans by the innate immune system. Nat Rev Microbiol 6:67–78
-
(2008)
Nat Rev Microbiol
, vol.6
, pp. 67-78
-
-
Netea, M.G.1
-
5
-
-
77950500877
-
Anti-Aspergillus human host defence relies on type 1 T helper (Th1), rather than type 17 T helper (Th17), cellular immunity
-
COI: 1:CAS:528:DC%2BC3cXlt1Kntr0%3D, PID: 20002791
-
Chai LY et al (2010) Anti-Aspergillus human host defence relies on type 1 T helper (Th1), rather than type 17 T helper (Th17), cellular immunity. Immunology 130:46–54
-
(2010)
Immunology
, vol.130
, pp. 46-54
-
-
Chai, L.Y.1
-
6
-
-
77953289487
-
Dectin-2 recognition of alpha-mannans and induction of Th17 cell differentiation is essential for host defense against Candida albicans
-
COI: 1:CAS:528:DC%2BC3cXnt1Kls7Y%3D, PID: 20493731
-
Saijo S et al (2010) Dectin-2 recognition of alpha-mannans and induction of Th17 cell differentiation is essential for host defense against Candida albicans. Immunity 32:681–691
-
(2010)
Immunity
, vol.32
, pp. 681-691
-
-
Saijo, S.1
-
7
-
-
84894245468
-
Role of Dectin-2 for host defense against systemic infection with Candida glabrata
-
PID: 24343653
-
Ifrim DC et al (2014) Role of Dectin-2 for host defense against systemic infection with Candida glabrata. Infect Immun 82:1064–1073
-
(2014)
Infect Immun
, vol.82
, pp. 1064-1073
-
-
Ifrim, D.C.1
-
8
-
-
84882714745
-
C-type lectin receptors Dectin-3 and Dectin-2 form a heterodimeric pattern-recognition receptor for host defense against fungal infection
-
COI: 1:CAS:528:DC%2BC3sXht1SrsrrI, PID: 23911656
-
Zhu LL et al (2013) C-type lectin receptors Dectin-3 and Dectin-2 form a heterodimeric pattern-recognition receptor for host defense against fungal infection. Immunity 39:324–334
-
(2013)
Immunity
, vol.39
, pp. 324-334
-
-
Zhu, L.L.1
-
9
-
-
84882793929
-
Dectin-2 is predominately macrophage restricted and exhibits conspicuous expression during Aspergillus fumigatus invasion in human lung
-
COI: 1:CAS:528:DC%2BC3sXhsFenu7fE, PID: 23928558
-
Sun H et al (2013) Dectin-2 is predominately macrophage restricted and exhibits conspicuous expression during Aspergillus fumigatus invasion in human lung. Cell Immunol 284:60–67
-
(2013)
Cell Immunol
, vol.284
, pp. 60-67
-
-
Sun, H.1
-
10
-
-
33749143645
-
Specific recognition of Candida albicans by macrophages requires galectin-3 to discriminate Saccharomyces cerevisiae and needs association with TLR2 for signaling
-
COI: 1:CAS:528:DC%2BD28Xps1Gnsrw%3D, PID: 16982907
-
Jouault T et al (2006) Specific recognition of Candida albicans by macrophages requires galectin-3 to discriminate Saccharomyces cerevisiae and needs association with TLR2 for signaling. J Immunol 177:4679–4687
-
(2006)
J Immunol
, vol.177
, pp. 4679-4687
-
-
Jouault, T.1
-
11
-
-
84880308194
-
Galectin-3 plays an important role in protection against disseminated candidiasis
-
COI: 1:CAS:528:DC%2BC3sXhtFamsbbE, PID: 23488971
-
Linden JR et al (2013) Galectin-3 plays an important role in protection against disseminated candidiasis. Med Mycol 51:641–651
-
(2013)
Med Mycol
, vol.51
, pp. 641-651
-
-
Linden, J.R.1
-
12
-
-
47249089542
-
The macrophage-inducible C-type lectin, mincle, is an essential component of the innate immune response to Candida albicans
-
COI: 1:CAS:528:DC%2BD1cXmtVanu74%3D, PID: 18490740
-
Wells CA et al (2008) The macrophage-inducible C-type lectin, mincle, is an essential component of the innate immune response to Candida albicans. J Immunol 180:7404–7413
-
(2008)
J Immunol
, vol.180
, pp. 7404-7413
-
-
Wells, C.A.1
-
13
-
-
50649113319
-
Dendritic cell interaction with Candida albicans critically depends on N-linked mannan
-
COI: 1:CAS:528:DC%2BD1cXotlOhs7k%3D, PID: 18482990
-
Cambi A et al (2008) Dendritic cell interaction with Candida albicans critically depends on N-linked mannan. J Biol Chem 283:20590–20599
-
(2008)
J Biol Chem
, vol.283
, pp. 20590-20599
-
-
Cambi, A.1
-
14
-
-
6344268943
-
Dendritic cell-specific intercellular adhesion molecule 3-grabbing nonintegrin mediates binding and internalization of Aspergillus fumigatus conidia by dendritic cells and macrophages
-
COI: 1:CAS:528:DC%2BD2cXos1Ghs78%3D, PID: 15494514
-
Serrano-Gomez D et al (2004) Dendritic cell-specific intercellular adhesion molecule 3-grabbing nonintegrin mediates binding and internalization of Aspergillus fumigatus conidia by dendritic cells and macrophages. J Immunol 173:5635–5643
-
(2004)
J Immunol
, vol.173
, pp. 5635-5643
-
-
Serrano-Gomez, D.1
-
15
-
-
3242660135
-
The potential role of mannan-binding lectin in the clearance of self-components including immune complexes
-
COI: 1:CAS:528:DC%2BD2cXms1aiurg%3D, PID: 15238070
-
Saevarsdottir S et al (2004) The potential role of mannan-binding lectin in the clearance of self-components including immune complexes. Scand J Immunol 60:23–29
-
(2004)
Scand J Immunol
, vol.60
, pp. 23-29
-
-
Saevarsdottir, S.1
-
16
-
-
0037025994
-
Dectin-1 is a major beta-glucan receptor on macrophages
-
COI: 1:CAS:528:DC%2BD38XmtVOhtb4%3D, PID: 12163569
-
Brown GD et al (2002) Dectin-1 is a major beta-glucan receptor on macrophages. J Exp Med 196:407–412
-
(2002)
J Exp Med
, vol.196
, pp. 407-412
-
-
Brown, G.D.1
-
17
-
-
79955537225
-
Activation of the innate immune receptor Dectin-1 upon formation of a 'phagocytic synapse'
-
COI: 1:CAS:528:DC%2BC3MXlsVSitbo%3D, PID: 21525931
-
Goodridge HS et al (2011) Activation of the innate immune receptor Dectin-1 upon formation of a 'phagocytic synapse'. Nature 472:471–475
-
(2011)
Nature
, vol.472
, pp. 471-475
-
-
Goodridge, H.S.1
-
18
-
-
84878077706
-
Aspergillus fumigatus-induced IL-22 is not restricted to a specific Th cell subset and is dependent on complement receptor 3
-
COI: 1:CAS:528:DC%2BC3sXnvVWrt7w%3D, PID: 23645883
-
Gresnigt MS et al (2013) Aspergillus fumigatus-induced IL-22 is not restricted to a specific Th cell subset and is dependent on complement receptor 3. J Immunol 190:5629–5639
-
(2013)
J Immunol
, vol.190
, pp. 5629-5639
-
-
Gresnigt, M.S.1
-
19
-
-
84923108179
-
Pattern recognition pathways leading to a Th2 cytokine bias in ABPA patients. Clin Exp Allergy
-
Becker KL et al. (2014) Pattern recognition pathways leading to a Th2 cytokine bias in ABPA patients. Clin Exp Allergy. doi:10.1111/cea.12354.
-
(2014)
doi:10.1111/cea.12354
-
-
Becker, K.L.1
-
20
-
-
84856551310
-
Persistence versus escape: Aspergillus terreus and Aspergillus fumigatus employ different strategies during interactions with macrophages
-
COI: 1:CAS:528:DC%2BC38Xis12hu7w%3D, PID: 22319619
-
Slesiona S et al (2012) Persistence versus escape: Aspergillus terreus and Aspergillus fumigatus employ different strategies during interactions with macrophages. PLoS One 7:e31223
-
(2012)
PLoS One
, vol.7
, pp. e31223
-
-
Slesiona, S.1
-
21
-
-
37349107599
-
The beta-glucan receptor dectin-1 recognizes specific morphologies of Aspergillus fumigatus
-
Steele C et al. (2005) The beta-glucan receptor dectin-1 recognizes specific morphologies of Aspergillus fumigatus. In PLoS Pathog 1(4): e42. doi:10.1371/journal.ppat.0010042
-
(2005)
In PLoS Pathog
, vol.1
, Issue.4
, pp. e42
-
-
Steele, C.1
-
22
-
-
80855127745
-
Neutrophils produce interleukin 17A (IL-17A) in a dectin-1- and IL-23-dependent manner during invasive fungal infection
-
COI: 1:CAS:528:DC%2BC3MXhtlOkur7K, PID: 21807912
-
Werner JL et al (2011) Neutrophils produce interleukin 17A (IL-17A) in a dectin-1- and IL-23-dependent manner during invasive fungal infection. Infect Immun 79:3966–3977
-
(2011)
Infect Immun
, vol.79
, pp. 3966-3977
-
-
Werner, J.L.1
-
23
-
-
84866542660
-
The beta-glucan receptor dectin-1 promotes lung immunopathology during fungal allergy via IL-22
-
COI: 1:CAS:528:DC%2BC38XhtlOmu7%2FN, PID: 22933634
-
Lilly LM et al (2012) The beta-glucan receptor dectin-1 promotes lung immunopathology during fungal allergy via IL-22. J Immunol 189:3653–3660
-
(2012)
J Immunol
, vol.189
, pp. 3653-3660
-
-
Lilly, L.M.1
-
24
-
-
84880668491
-
Corticosteroids block autophagy protein recruitment in Aspergillus fumigatus phagosomes via targeting dectin-1/Syk kinase signaling
-
COI: 1:CAS:528:DC%2BC3sXhtFCjsr7F, PID: 23817424
-
Kyrmizi I et al (2013) Corticosteroids block autophagy protein recruitment in Aspergillus fumigatus phagosomes via targeting dectin-1/Syk kinase signaling. J Immunol 191:1287–1299
-
(2013)
J Immunol
, vol.191
, pp. 1287-1299
-
-
Kyrmizi, I.1
-
25
-
-
56149097181
-
TH17 cells mediate steroid-resistant airway inflammation and airway hyperresponsiveness in mice
-
COI: 1:CAS:528:DC%2BD1cXhtVKntb%2FK, PID: 18768865
-
McKinley L et al (2008) TH17 cells mediate steroid-resistant airway inflammation and airway hyperresponsiveness in mice. J Immunol 181:4089–4097
-
(2008)
J Immunol
, vol.181
, pp. 4089-4097
-
-
McKinley, L.1
-
26
-
-
84883427626
-
Human genetic susceptibility to invasive aspergillosis
-
COI: 1:CAS:528:DC%2BC3sXhsVensrrK, PID: 23950708
-
Cunha C et al (2013) Human genetic susceptibility to invasive aspergillosis. PLoS Pathog 9:e1003434
-
(2013)
PLoS Pathog
, vol.9
, pp. e1003434
-
-
Cunha, C.1
-
27
-
-
84878699314
-
Genetic susceptibility to Candida infections
-
COI: 1:CAS:528:DC%2BC3sXos12ntb4%3D, PID: 23629947
-
Smeekens SP et al (2013) Genetic susceptibility to Candida infections. EMBO Mol Med 5:805–813
-
(2013)
EMBO Mol Med
, vol.5
, pp. 805-813
-
-
Smeekens, S.P.1
-
28
-
-
10744221913
-
The contribution of the Toll-like/IL-1 receptor superfamily to innate and adaptive immunity to fungal pathogens in vivo
-
COI: 1:CAS:528:DC%2BD2cXhsVCrtb0%3D, PID: 14978111
-
Bellocchio S et al (2004) The contribution of the Toll-like/IL-1 receptor superfamily to innate and adaptive immunity to fungal pathogens in vivo. J Immunol 172:3059–3069
-
(2004)
J Immunol
, vol.172
, pp. 3059-3069
-
-
Bellocchio, S.1
-
29
-
-
1542409922
-
Toll-like receptor 2 suppresses immunity against Candida albicans through induction of IL-10 and regulatory T cells
-
COI: 1:CAS:528:DC%2BD2cXhvVKis7k%3D, PID: 15004175
-
Netea MG et al (2004) Toll-like receptor 2 suppresses immunity against Candida albicans through induction of IL-10 and regulatory T cells. J Immunol 172:3712–3718
-
(2004)
J Immunol
, vol.172
, pp. 3712-3718
-
-
Netea, M.G.1
-
30
-
-
0037094116
-
The role of toll-like receptor (TLR) 2 and TLR4 in the host defense against disseminated candidiasis
-
COI: 1:CAS:528:DC%2BD38XksVCjtbY%3D, PID: 11992285
-
Netea MG et al (2002) The role of toll-like receptor (TLR) 2 and TLR4 in the host defense against disseminated candidiasis. J Infect Dis 185:1483–1489
-
(2002)
J Infect Dis
, vol.185
, pp. 1483-1489
-
-
Netea, M.G.1
-
31
-
-
54949144335
-
Toll-like receptor 4 polymorphisms and aspergillosis in stem-cell transplantation
-
COI: 1:CAS:528:DC%2BD1cXht12nu73I, PID: 18946062
-
Bochud PY et al (2008) Toll-like receptor 4 polymorphisms and aspergillosis in stem-cell transplantation. N Engl J Med 359:1766–1777
-
(2008)
N Engl J Med
, vol.359
, pp. 1766-1777
-
-
Bochud, P.Y.1
-
32
-
-
23944515364
-
Involvement of toll-like receptor 2 in experimental invasive pulmonary aspergillosis
-
COI: 1:CAS:528:DC%2BD2MXps1GksLg%3D, PID: 16113258
-
Balloy V et al (2005) Involvement of toll-like receptor 2 in experimental invasive pulmonary aspergillosis. Infect Immun 73:5420–5425
-
(2005)
Infect Immun
, vol.73
, pp. 5420-5425
-
-
Balloy, V.1
-
33
-
-
79551469099
-
The L412F variant of Toll-like receptor 3 (TLR3) is associated with cutaneous candidiasis, increased susceptibility to cytomegalovirus, and autoimmunity
-
COI: 1:CAS:528:DC%2BC3MXhtlSqu70%3D, PID: 21093032
-
Nahum A et al (2011) The L412F variant of Toll-like receptor 3 (TLR3) is associated with cutaneous candidiasis, increased susceptibility to cytomegalovirus, and autoimmunity. J Allergy Clin Immunol 127:528–531
-
(2011)
J Allergy Clin Immunol
, vol.127
, pp. 528-531
-
-
Nahum, A.1
-
34
-
-
84856267624
-
TLR3 essentially promotes protective class I-restricted memory CD8(+) T-cell responses to Aspergillus fumigatus in hematopoietic transplanted patients
-
COI: 1:CAS:528:DC%2BC38XitVKqt7c%3D, PID: 22147891
-
Carvalho A et al (2012) TLR3 essentially promotes protective class I-restricted memory CD8(+) T-cell responses to Aspergillus fumigatus in hematopoietic transplanted patients. Blood 119:967–977
-
(2012)
Blood
, vol.119
, pp. 967-977
-
-
Carvalho, A.1
-
35
-
-
84901313667
-
Fungal chitin dampens inflammation through IL-10 induction mediated by NOD2 and TLR9 activation
-
PID: 24722226
-
Wagener J et al (2014) Fungal chitin dampens inflammation through IL-10 induction mediated by NOD2 and TLR9 activation. PLoS Pathog 10:e1004050
-
(2014)
PLoS Pathog
, vol.10
, pp. e1004050
-
-
Wagener, J.1
-
36
-
-
69549108298
-
Dectin-2 is a Syk-coupled pattern recognition receptor crucial for Th17 responses to fungal infection
-
COI: 1:CAS:528:DC%2BD1MXhtVyitLbI, PID: 19703985
-
Robinson MJ et al (2009) Dectin-2 is a Syk-coupled pattern recognition receptor crucial for Th17 responses to fungal infection. J Exp Med 206:2037–2051
-
(2009)
J Exp Med
, vol.206
, pp. 2037-2051
-
-
Robinson, M.J.1
-
37
-
-
67349271142
-
Syk kinase signalling couples to the Nlrp3 inflammasome for anti-fungal host defence
-
COI: 1:CAS:528:DC%2BD1MXjvV2ksLs%3D, PID: 19339971
-
Gross O et al (2009) Syk kinase signalling couples to the Nlrp3 inflammasome for anti-fungal host defence. Nature 459:433–436
-
(2009)
Nature
, vol.459
, pp. 433-436
-
-
Gross, O.1
-
38
-
-
65549154784
-
An essential role for the NLRP3 inflammasome in host defense against the human fungal pathogen Candida albicans
-
COI: 1:CAS:528:DC%2BD1MXosVClu7c%3D, PID: 19454352
-
Hise AG et al (2009) An essential role for the NLRP3 inflammasome in host defense against the human fungal pathogen Candida albicans. Cell Host Microbe 5:487–497
-
(2009)
Cell Host Microbe
, vol.5
, pp. 487-497
-
-
Hise, A.G.1
-
39
-
-
77954958602
-
Aspergillus fumigatus stimulates the NLRP3 inflammasome through a pathway requiring ROS production and the Syk tyrosine kinase
-
PID: 20368800
-
Said-Sadier N et al (2010) Aspergillus fumigatus stimulates the NLRP3 inflammasome through a pathway requiring ROS production and the Syk tyrosine kinase. PLoS One 5:e10008
-
(2010)
PLoS One
, vol.5
, pp. e10008
-
-
Said-Sadier, N.1
-
40
-
-
77956583415
-
A biphasic innate immune MAPK response discriminates between the yeast and hyphal forms of Candida albicans in epithelial cells
-
COI: 1:CAS:528:DC%2BC3cXhtFGjsbfF, PID: 20833374
-
Moyes DL et al (2010) A biphasic innate immune MAPK response discriminates between the yeast and hyphal forms of Candida albicans in epithelial cells. Cell Host Microbe 8:225–235
-
(2010)
Cell Host Microbe
, vol.8
, pp. 225-235
-
-
Moyes, D.L.1
-
41
-
-
46149097893
-
Phagocytosis of Aspergillus fumigatus conidia by primary nasal epithelial cells in vitro
-
PID: 18564423
-
Botterel F et al (2008) Phagocytosis of Aspergillus fumigatus conidia by primary nasal epithelial cells in vitro. BMC Microbiol 8:97
-
(2008)
BMC Microbiol
, vol.8
, pp. 97
-
-
Botterel, F.1
-
42
-
-
84902136675
-
Proteomic characterization of human proinflammatory M1 and anti-inflammatory M2 macrophages and their response to Candida albicans
-
COI: 1:CAS:528:DC%2BC2cXotlygtbs%3D, PID: 24687989
-
Reales-Calderon JA et al (2014) Proteomic characterization of human proinflammatory M1 and anti-inflammatory M2 macrophages and their response to Candida albicans. Proteomics 14:1503–1518
-
(2014)
Proteomics
, vol.14
, pp. 1503-1518
-
-
Reales-Calderon, J.A.1
-
43
-
-
84890020963
-
CX3CR1-dependent renal macrophage survival promotes Candida control and host survival
-
COI: 1:CAS:528:DC%2BC3sXhvFSqtbfM, PID: 24177428
-
Lionakis MS et al (2013) CX3CR1-dependent renal macrophage survival promotes Candida control and host survival. J Clin Invest 123:5035–5051
-
(2013)
J Clin Invest
, vol.123
, pp. 5035-5051
-
-
Lionakis, M.S.1
-
44
-
-
84891365225
-
Inflammatory monocytes mediate early and organ-specific innate defense during systemic candidiasis
-
COI: 1:CAS:528:DC%2BC3sXhvFeqt7%2FI, PID: 23922372
-
Ngo LY et al (2014) Inflammatory monocytes mediate early and organ-specific innate defense during systemic candidiasis. J Infect Dis 209:109–119
-
(2014)
J Infect Dis
, vol.209
, pp. 109-119
-
-
Ngo, L.Y.1
-
45
-
-
71749100858
-
Inflammatory monocytes facilitate adaptive CD4 T cell responses during respiratory fungal infection
-
COI: 1:CAS:528:DC%2BD1MXhsF2gs73K, PID: 19917501
-
Hohl TM et al (2009) Inflammatory monocytes facilitate adaptive CD4 T cell responses during respiratory fungal infection. Cell Host Microbe 6:470–481
-
(2009)
Cell Host Microbe
, vol.6
, pp. 470-481
-
-
Hohl, T.M.1
-
46
-
-
84895754889
-
Inflammatory monocytes orchestrate innate antifungal immunity in the lung
-
PID: 24586155
-
Espinosa V et al (2014) Inflammatory monocytes orchestrate innate antifungal immunity in the lung. PLoS Pathog 10:e1003940
-
(2014)
PLoS Pathog
, vol.10
, pp. e1003940
-
-
Espinosa, V.1
-
47
-
-
69149089714
-
Essential role for neutrophils but not alveolar macrophages at early time points following Aspergillus fumigatus infection
-
COI: 1:CAS:528:DC%2BD1MXhtV2gs7bO, PID: 19591573
-
Mircescu MM et al (2009) Essential role for neutrophils but not alveolar macrophages at early time points following Aspergillus fumigatus infection. J Infect Dis 200:647–656
-
(2009)
J Infect Dis
, vol.200
, pp. 647-656
-
-
Mircescu, M.M.1
-
48
-
-
84859387081
-
Neutrophil function: from mechanisms to disease
-
COI: 1:CAS:528:DC%2BC38XmsVKmur0%3D, PID: 22224774
-
Amulic B et al (2012) Neutrophil function: from mechanisms to disease. Annu Rev Immunol 30:459–489
-
(2012)
Annu Rev Immunol
, vol.30
, pp. 459-489
-
-
Amulic, B.1
-
49
-
-
34248188692
-
Human polymorphonuclear leukocytes inhibit Aspergillus fumigatus conidial growth by lactoferrin-mediated iron depletion
-
COI: 1:CAS:528:DC%2BD2sXksl2nu7o%3D, PID: 17475866
-
Zarember KA et al (2007) Human polymorphonuclear leukocytes inhibit Aspergillus fumigatus conidial growth by lactoferrin-mediated iron depletion. J Immunol 178:6367–6373
-
(2007)
J Immunol
, vol.178
, pp. 6367-6373
-
-
Zarember, K.A.1
-
50
-
-
77958091269
-
NETs formed by human neutrophils inhibit growth of the pathogenic mold Aspergillus fumigatus
-
COI: 1:CAS:528:DC%2BC3cXhtlCgs73L, PID: 20603224
-
McCormick A et al (2010) NETs formed by human neutrophils inhibit growth of the pathogenic mold Aspergillus fumigatus. Microbes Infect 12:928–936
-
(2010)
Microbes Infect
, vol.12
, pp. 928-936
-
-
McCormick, A.1
-
51
-
-
84856598960
-
Killing by neutrophil extracellular traps: fact or folklore?
-
COI: 1:CAS:528:DC%2BC38XisFOit7g%3D, PID: 22210873
-
Menegazzi R et al (2012) Killing by neutrophil extracellular traps: fact or folklore? Blood 119:1214–1216
-
(2012)
Blood
, vol.119
, pp. 1214-1216
-
-
Menegazzi, R.1
-
52
-
-
77954161132
-
Production of extracellular traps against Aspergillus fumigatus in vitro and in infected lung tissue is dependent on invading neutrophils and influenced by hydrophobin RodA
-
PID: 20442864
-
Bruns S et al (2010) Production of extracellular traps against Aspergillus fumigatus in vitro and in infected lung tissue is dependent on invading neutrophils and influenced by hydrophobin RodA. PLoS Pathog 6:e1000873
-
(2010)
PLoS Pathog
, vol.6
, pp. e1000873
-
-
Bruns, S.1
-
53
-
-
79955596509
-
Restoration of anti-Aspergillus defense by neutrophil extracellular traps in human chronic granulomatous disease after gene therapy is calprotectin-dependent
-
PID: 21376380
-
Bianchi M et al (2011) Restoration of anti-Aspergillus defense by neutrophil extracellular traps in human chronic granulomatous disease after gene therapy is calprotectin-dependent. J Allergy Clin Immunol 127(1243–52):e7
-
(2011)
J Allergy Clin Immunol
, vol.127
, Issue.1243-52
, pp. e7
-
-
Bianchi, M.1
-
54
-
-
73649099522
-
Neutrophil extracellular traps contain calprotectin, a cytosolic protein complex involved in host defense against Candida albicans
-
PID: 19876394
-
Urban CF et al (2009) Neutrophil extracellular traps contain calprotectin, a cytosolic protein complex involved in host defense against Candida albicans. PLoS Pathog 5:e1000639
-
(2009)
PLoS Pathog
, vol.5
, pp. e1000639
-
-
Urban, C.F.1
-
55
-
-
84876008656
-
Monocyte- and macrophage-targeted NADPH oxidase mediates antifungal host defense and regulation of acute inflammation in mice
-
COI: 1:CAS:528:DC%2BC3sXlsVCltr0%3D, PID: 23509361
-
Grimm MJ et al (2013) Monocyte- and macrophage-targeted NADPH oxidase mediates antifungal host defense and regulation of acute inflammation in mice. J Immunol 190:4175–4184
-
(2013)
J Immunol
, vol.190
, pp. 4175-4184
-
-
Grimm, M.J.1
-
56
-
-
84895819096
-
IL-1 receptor blockade restores autophagy and reduces inflammation in chronic granulomatous disease in mice and in humans
-
PID: 24550444
-
de Luca A et al (2014) IL-1 receptor blockade restores autophagy and reduces inflammation in chronic granulomatous disease in mice and in humans. Proc Natl Acad Sci U S A 111:3526–3531
-
(2014)
Proc Natl Acad Sci U S A
, vol.111
, pp. 3526-3531
-
-
de Luca, A.1
-
57
-
-
84900857988
-
Autophagy is redundant for the host defense against systemic Candida albicans infections
-
COI: 1:CAS:528:DC%2BC3sXhslKqtLjJ, PID: 24202731
-
Smeekens SP et al (2014) Autophagy is redundant for the host defense against systemic Candida albicans infections. Eur J Clin Microbiol Infect Dis 33:711–722
-
(2014)
Eur J Clin Microbiol Infect Dis
, vol.33
, pp. 711-722
-
-
Smeekens, S.P.1
-
58
-
-
84867605302
-
Macrophage autophagy in immunity to Cryptococcus neoformans and Candida albicans
-
COI: 1:CAS:528:DC%2BC38Xht1OrsLzO, PID: 22710871
-
Nicola AM et al (2012) Macrophage autophagy in immunity to Cryptococcus neoformans and Candida albicans. Infect Immun 80:3065–3076
-
(2012)
Infect Immun
, vol.80
, pp. 3065-3076
-
-
Nicola, A.M.1
-
59
-
-
84904910793
-
Two independent killing mechanisms of Candida albicans by human neutrophils: evidence from innate immunity defects
-
COI: 1:CAS:528:DC%2BC2cXht1Ggu7zN, PID: 24948657
-
Gazendam RP et al (2014) Two independent killing mechanisms of Candida albicans by human neutrophils: evidence from innate immunity defects. Blood 124(4):590–597
-
(2014)
Blood
, vol.124
, Issue.4
, pp. 590-597
-
-
Gazendam, R.P.1
-
60
-
-
84892827750
-
Activation of neutrophils by autocrine IL-17A-IL-17RC interactions during fungal infection is regulated by IL-6, IL-23, RORgammat and dectin-2
-
COI: 1:CAS:528:DC%2BC3sXhvFOltLvN, PID: 24362892
-
Taylor PR et al (2014) Activation of neutrophils by autocrine IL-17A-IL-17RC interactions during fungal infection is regulated by IL-6, IL-23, RORgammat and dectin-2. Nat Immunol 15:143–151
-
(2014)
Nat Immunol
, vol.15
, pp. 143-151
-
-
Taylor, P.R.1
-
61
-
-
84894270649
-
Role of neutrophils in IL-17-dependent immunity to mucosal candidiasis
-
COI: 1:CAS:528:DC%2BC2cXitFWmsrw%3D, PID: 24442441
-
Huppler AR et al (2014) Role of neutrophils in IL-17-dependent immunity to mucosal candidiasis. J Immunol 192:1745–1752
-
(2014)
J Immunol
, vol.192
, pp. 1745-1752
-
-
Huppler, A.R.1
-
62
-
-
84866650842
-
The acute neutrophil response mediated by S100 alarmins during vaginal Candida infections is independent of the Th17-pathway
-
COI: 1:CAS:528:DC%2BC38XhsVGhtL3L, PID: 23050010
-
Yano J et al (2012) The acute neutrophil response mediated by S100 alarmins during vaginal Candida infections is independent of the Th17-pathway. PLoS One 7:e46311
-
(2012)
PLoS One
, vol.7
, pp. e46311
-
-
Yano, J.1
-
63
-
-
84897558259
-
In vitro effects of Candida albicans and Aspergillus fumigatus on dendritic cells and the role of beta glucan in this effect
-
PID: 24595999
-
Fidan I et al (2014) In vitro effects of Candida albicans and Aspergillus fumigatus on dendritic cells and the role of beta glucan in this effect. Adv Clin Exp Med 23:17–24
-
(2014)
Adv Clin Exp Med
, vol.23
, pp. 17-24
-
-
Fidan, I.1
-
64
-
-
79956314118
-
Recognition of yeast nucleic acids triggers a host-protective type I interferon response
-
COI: 1:CAS:528:DC%2BC3MXnvF2gsbs%3D, PID: 21480215
-
Biondo C et al (2011) Recognition of yeast nucleic acids triggers a host-protective type I interferon response. Eur J Immunol 41:1969–1979
-
(2011)
Eur J Immunol
, vol.41
, pp. 1969-1979
-
-
Biondo, C.1
-
65
-
-
84879602096
-
Interferon-beta production via Dectin-1-Syk-IRF5 signaling in dendritic cells is crucial for immunity to C. albicans
-
PID: 23770228
-
del Fresno C et al (2013) Interferon-beta production via Dectin-1-Syk-IRF5 signaling in dendritic cells is crucial for immunity to C. albicans. Immunity 38:1176–1186
-
(2013)
Immunity
, vol.38
, pp. 1176-1186
-
-
del Fresno, C.1
-
66
-
-
79952774989
-
Conventional dendritic cells mount a type I IFN response against Candida spp. requiring novel phagosomal TLR7-mediated IFN-beta signaling
-
COI: 1:CAS:528:DC%2BC3MXhvFCjsL0%3D, PID: 21282509
-
Bourgeois C et al (2011) Conventional dendritic cells mount a type I IFN response against Candida spp. requiring novel phagosomal TLR7-mediated IFN-beta signaling. J Immunol 186:3104–3112
-
(2011)
J Immunol
, vol.186
, pp. 3104-3112
-
-
Bourgeois, C.1
-
67
-
-
77249102130
-
Intranasally delivered siRNA targeting PI3K/Akt/mTOR inflammatory pathways protects from aspergillosis
-
COI: 1:CAS:528:DC%2BC3cXhvVKjsr8%3D, PID: 19924119
-
Bonifazi P et al (2010) Intranasally delivered siRNA targeting PI3K/Akt/mTOR inflammatory pathways protects from aspergillosis. Mucosal Immunol 3:193–205
-
(2010)
Mucosal Immunol
, vol.3
, pp. 193-205
-
-
Bonifazi, P.1
-
68
-
-
79955082224
-
TNF-alpha from inflammatory dendritic cells (DCs) regulates lung IL-17A/IL-5 levels and neutrophilia versus eosinophilia during persistent fungal infection
-
COI: 1:CAS:528:DC%2BC3MXkslOmtbg%3D, PID: 21402950
-
Fei M et al (2011) TNF-alpha from inflammatory dendritic cells (DCs) regulates lung IL-17A/IL-5 levels and neutrophilia versus eosinophilia during persistent fungal infection. Proc Natl Acad Sci U S A 108:5360–5365
-
(2011)
Proc Natl Acad Sci U S A
, vol.108
, pp. 5360-5365
-
-
Fei, M.1
-
69
-
-
79956145147
-
A nonredundant role for plasmacytoid dendritic cells in host defense against the human fungal pathogen Aspergillus fumigatus
-
COI: 1:CAS:528:DC%2BC3MXmtFaisro%3D, PID: 21575912
-
Ramirez-Ortiz ZG et al (2011) A nonredundant role for plasmacytoid dendritic cells in host defense against the human fungal pathogen Aspergillus fumigatus. Cell Host Microbe 9:415–424
-
(2011)
Cell Host Microbe
, vol.9
, pp. 415-424
-
-
Ramirez-Ortiz, Z.G.1
-
70
-
-
33646940680
-
Role for dendritic cells in immunoregulation during experimental vaginal candidiasis
-
COI: 1:CAS:528:DC%2BD28Xlt1Cgtb8%3D, PID: 16714548
-
LeBlanc DM et al (2006) Role for dendritic cells in immunoregulation during experimental vaginal candidiasis. Infect Immun 74:3213–3221
-
(2006)
Infect Immun
, vol.74
, pp. 3213-3221
-
-
LeBlanc, D.M.1
-
71
-
-
64649083975
-
The macrophage mannose receptor induces IL-17 in response to Candida albicans
-
PID: 19380112
-
van de Veerdonk FL et al (2009) The macrophage mannose receptor induces IL-17 in response to Candida albicans. Cell Host Microbe 5:329–340
-
(2009)
Cell Host Microbe
, vol.5
, pp. 329-340
-
-
van de Veerdonk, F.L.1
-
72
-
-
63049138176
-
Th17 cells and IL-17 receptor signaling are essential for mucosal host defense against oral candidiasis
-
COI: 1:CAS:528:DC%2BD1MXit1Kjtbc%3D, PID: 19204111
-
Conti HR et al (2009) Th17 cells and IL-17 receptor signaling are essential for mucosal host defense against oral candidiasis. J Exp Med 206:299–311
-
(2009)
J Exp Med
, vol.206
, pp. 299-311
-
-
Conti, H.R.1
-
73
-
-
34548317417
-
Dominant-negative mutations in the DNA-binding domain of STAT3 cause hyper-IgE syndrome
-
COI: 1:CAS:528:DC%2BD2sXps12nsLc%3D, PID: 17676033
-
Minegishi Y et al (2007) Dominant-negative mutations in the DNA-binding domain of STAT3 cause hyper-IgE syndrome. Nature 448:1058–1062
-
(2007)
Nature
, vol.448
, pp. 1058-1062
-
-
Minegishi, Y.1
-
74
-
-
79960094057
-
STAT1 mutations in autosomal dominant chronic mucocutaneous candidiasis
-
PID: 21714643
-
van de Veerdonk FL et al (2011) STAT1 mutations in autosomal dominant chronic mucocutaneous candidiasis. N Engl J Med 365:54–61
-
(2011)
N Engl J Med
, vol.365
, pp. 54-61
-
-
van de Veerdonk, F.L.1
-
75
-
-
84872171847
-
Cutting edge: IL-17-secreting innate lymphoid cells are essential for host defense against fungal infection
-
COI: 1:CAS:528:DC%2BC3sXjtlGqtg%3D%3D, PID: 23255360
-
Gladiator A et al (2013) Cutting edge: IL-17-secreting innate lymphoid cells are essential for host defense against fungal infection. J Immunol 190:521–525
-
(2013)
J Immunol
, vol.190
, pp. 521-525
-
-
Gladiator, A.1
-
76
-
-
84890824171
-
Candida albicans airway exposure primes the lung innate immune response against Pseudomonas aeruginosa infection through innate lymphoid cell recruitment and interleukin-22-associated mucosal response
-
PID: 24166952
-
Mear JB et al (2014) Candida albicans airway exposure primes the lung innate immune response against Pseudomonas aeruginosa infection through innate lymphoid cell recruitment and interleukin-22-associated mucosal response. Infect Immun 82:306–315
-
(2014)
Infect Immun
, vol.82
, pp. 306-315
-
-
Mear, J.B.1
-
77
-
-
84882625503
-
IL-33-responsive innate lymphoid cells are an important source of IL-13 in chronic rhinosinusitis with nasal polyps
-
COI: 1:CAS:528:DC%2BC3sXhsFeqsb7P, PID: 23805875
-
Shaw JL et al (2013) IL-33-responsive innate lymphoid cells are an important source of IL-13 in chronic rhinosinusitis with nasal polyps. Am J Respir Crit Care Med 188:432–439
-
(2013)
Am J Respir Crit Care Med
, vol.188
, pp. 432-439
-
-
Shaw, J.L.1
-
78
-
-
84886262448
-
Invariant natural killer T cells recognize a fungal glycosphingolipid that can induce airway hyperreactivity
-
COI: 1:CAS:528:DC%2BC3sXhtlGltL7J, PID: 23995283
-
Albacker LA et al (2013) Invariant natural killer T cells recognize a fungal glycosphingolipid that can induce airway hyperreactivity. Nat Med 19:1297–1304
-
(2013)
Nat Med
, vol.19
, pp. 1297-1304
-
-
Albacker, L.A.1
-
79
-
-
84906093170
-
Differential role of NK cells against Candida albicans infection in immunocompetent or immunocompromised mice
-
COI: 1:CAS:528:DC%2BC2cXosFentLo%3D, PID: 24802993
-
Quintin J et al (2014) Differential role of NK cells against Candida albicans infection in immunocompetent or immunocompromised mice. Eur J Immunol 44(8):2405–2414
-
(2014)
Eur J Immunol
, vol.44
, Issue.8
, pp. 2405-2414
-
-
Quintin, J.1
-
80
-
-
84893259804
-
Human natural killer cells acting as phagocytes against Candida albicans and mounting an inflammatory response that modulates neutrophil antifungal activity
-
COI: 1:CAS:528:DC%2BC2cXhsFaktbo%3D, PID: 24163416
-
Voigt J et al (2014) Human natural killer cells acting as phagocytes against Candida albicans and mounting an inflammatory response that modulates neutrophil antifungal activity. J Infect Dis 209:616–626
-
(2014)
J Infect Dis
, vol.209
, pp. 616-626
-
-
Voigt, J.1
-
81
-
-
79751512293
-
Human natural killer cells exhibit direct activity against Aspergillus fumigatus hyphae, but not against resting conidia
-
COI: 1:CAS:528:DC%2BC3MXnsVWntg%3D%3D, PID: 21208932
-
Schmidt S et al (2011) Human natural killer cells exhibit direct activity against Aspergillus fumigatus hyphae, but not against resting conidia. J Infect Dis 203:430–435
-
(2011)
J Infect Dis
, vol.203
, pp. 430-435
-
-
Schmidt, S.1
-
82
-
-
80051657716
-
Human NK cells display important antifungal activity against Aspergillus fumigatus, which is directly mediated by IFN-gamma release
-
COI: 1:CAS:528:DC%2BC3MXptValsrc%3D, PID: 21697457
-
Bouzani M et al (2011) Human NK cells display important antifungal activity against Aspergillus fumigatus, which is directly mediated by IFN-gamma release. J Immunol 187:1369–1376
-
(2011)
J Immunol
, vol.187
, pp. 1369-1376
-
-
Bouzani, M.1
-
83
-
-
64249134349
-
Early NK cell-derived IFN-{gamma} is essential to host defense in neutropenic invasive aspergillosis
-
COI: 1:CAS:528:DC%2BD1MXjsVSjurs%3D, PID: 19299730
-
Park SJ et al (2009) Early NK cell-derived IFN-{gamma} is essential to host defense in neutropenic invasive aspergillosis. J Immunol 182:4306–4312
-
(2009)
J Immunol
, vol.182
, pp. 4306-4312
-
-
Park, S.J.1
-
84
-
-
0033955404
-
Adherence of platelets to Candida species in vivo
-
COI: 1:CAS:528:DC%2BD3cXotFOnug%3D%3D, PID: 10639419
-
Robert R et al (2000) Adherence of platelets to Candida species in vivo. Infect Immun 68:570–576
-
(2000)
Infect Immun
, vol.68
, pp. 570-576
-
-
Robert, R.1
-
85
-
-
84874183780
-
Antimicrobial activity of pure platelet-rich plasma against microorganisms isolated from oral cavity
-
COI: 1:CAS:528:DC%2BC3sXnsFaitLc%3D, PID: 23442413
-
Drago L et al (2013) Antimicrobial activity of pure platelet-rich plasma against microorganisms isolated from oral cavity. BMC Microbiol 13:47
-
(2013)
BMC Microbiol
, vol.13
, pp. 47
-
-
Drago, L.1
-
86
-
-
77349119806
-
Activation of platelets by Aspergillus fumigatus and potential role of platelets in the immunopathogenesis of aspergillosis
-
PID: 20008537
-
Rodland EK et al (2010) Activation of platelets by Aspergillus fumigatus and potential role of platelets in the immunopathogenesis of aspergillosis. Infect Immun 78:1269–1275
-
(2010)
Infect Immun
, vol.78
, pp. 1269-1275
-
-
Rodland, E.K.1
-
87
-
-
84873618860
-
Aspergillus fumigatus activates thrombocytes by secretion of soluble compounds
-
COI: 1:CAS:528:DC%2BC3sXit1Wmt7c%3D, PID: 23225903
-
Speth C et al (2013) Aspergillus fumigatus activates thrombocytes by secretion of soluble compounds. J Infect Dis 207:823–833
-
(2013)
J Infect Dis
, vol.207
, pp. 823-833
-
-
Speth, C.1
-
88
-
-
84865119423
-
Candida albicans infection affords protection against reinfection via functional reprogramming of monocytes
-
COI: 1:CAS:528:DC%2BC38Xht1SktL7N, PID: 22901542
-
Quintin J et al (2012) Candida albicans infection affords protection against reinfection via functional reprogramming of monocytes. Cell Host Microbe 12:223–232
-
(2012)
Cell Host Microbe
, vol.12
, pp. 223-232
-
-
Quintin, J.1
-
89
-
-
84895800375
-
Trained immunity or tolerance: opposing functional programs induced in human monocytes after engagement of various pattern recognition receptors
-
PID: 24521784
-
Ifrim DC et al (2014) Trained immunity or tolerance: opposing functional programs induced in human monocytes after engagement of various pattern recognition receptors. Clin Vaccine Immunol 21:534–545
-
(2014)
Clin Vaccine Immunol
, vol.21
, pp. 534-545
-
-
Ifrim, D.C.1
|