Hormesis and public health: Can glutathione depletion and mitochondrial dysfunction due to very low-dose chronic exposure to persistent organic pollutants be mitigated?

Journal of Epidemiology and Community Health

Volumn 69, Issue 3, 2015, Pages 294-300

  Lee, Duk Hee ^a,b   Jacobs, David R ^c  

^a Kyungpook National University School of Medicine   (South Korea)

^b Kyungpook National University   (South Korea)

^c UNIVERSITY OF MINNESOTA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANTIOXIDANT; ENDOCRINE DISRUPTOR; FOOD CHAIN; HEALTH IMPACT; HOMEOSTASIS; MITOCHONDRION; ORGANIC POLLUTANT; PERSISTENCE; POLLUTION EXPOSURE; PUBLIC HEALTH;

GLUTATHIONE; ORGANIC COMPOUND; POLLUTANT;

ADVERSE EFFECTS; AGE; ANIMAL; CHRONIC DISEASE; DEFICIENCY; DRUG EFFECTS; ENVIRONMENTAL EXPOSURE; HORMESIS; HUMAN; MITOCHONDRION; OXIDATIVE STRESS; PHYSIOLOGY; POLLUTANT; PREVENTION AND CONTROL; PUBLIC HEALTH;

AGE FACTORS; ANIMALS; CHRONIC DISEASE; ENVIRONMENTAL EXPOSURE; ENVIRONMENTAL POLLUTANTS; GLUTATHIONE; HORMESIS; HUMANS; MITOCHONDRIA; ORGANIC CHEMICALS; OXIDATIVE STRESS; PUBLIC HEALTH;

EID: 84924557923     PISSN: 0143005X     EISSN: 14702738     Source Type: Journal    
DOI: 10.1136/jech-2014-203861     Document Type: Review

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